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Researcher · Jamia Hamdard · India

Ashif Iqubal

Ashif Iqubal publishes mostly in Molecular Biology, Pathology and Forensic Medicine and Organic Chemistry, on topics such as Synthesis and biological activity, Computational Drug Discovery Methods and Chemotherapy-induced organ toxicity mitigation.

ORCID 0000-0003-4694-4851 OpenAlex
World rank
#571,578
of 7,710,434 ranked researchers
Rank in India
#6,974
of 280,097
Works
86
Citations
2.5k
Citations per work
29.5
h-index
26
i10-index 50Top 20% in Molecular Biology
Ashif Iqubal

What does Ashif Iqubal research?

Ashif Iqubal's work is mostly in Medicine (16%), Biochemistry, Genetics and Molecular Biology (12%), Chemistry (7%).

Everything else: 51 works · 59.6% of their workLife Sciences: 15 works · 17.5% of their workBiochemistry, Genetics and Molecular Biology: 10 works · 11.8% of their workMolecular Biology: 10 works · 11.8% of their workPharmacology, Toxicology and Pharmaceutics: 5 works · 5.7% of their workPharmaceutical Science: 5 works · 5.7% of their workHealth Sciences: 14 works · 16.3% of their workMedicine: 14 works · 16.3% of their workPathology and Forensic Medicine: 6 works · 7.5% of their workComplementary and alternative medicine: 4 works · 4.4% of their workOncology: 4 works · 4.4% of their workPhysical Sciences: 6 works · 6.6% of their workChemistry: 6 works · 6.6% of their workOrganic Chemistry: 6 works · 6.6% of their workTHEIR WORKAshif Iqubal86works
Life Sciences18%Physical Sciences7%Health Sciences16%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else5159.6%
    1. Biochemistry, Genetics and Molecular Biology1011.8%
    2. Pharmacology, Toxicology and Pharmaceutics55.7%
    1. Medicine1416.3%
    1. Chemistry66.6%
  1. 1Molecular Biology12% of their works
  2. 2Pathology and Forensic Medicine8% of their works
  3. 3Organic Chemistry7% of their works
  4. 4Pharmaceutical Science6% of their works
  5. 5Oncology4% of their works
  6. 6Complementary and alternative medicine4% of their works

Research topics

Which keywords describe Ashif Iqubal's research?

The keywords of their largest research topics: Oxidative Stress, Medicinal Chemistry, Neuroinflammation, Neurodegeneration, Cytokines, Microglia, Anticancer Agents and Heterocyclic Compounds.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Oxidative Stress203 topics
  2. Medicinal Chemistry182 topics
  3. Neuroinflammation173 topics
  4. Neurodegeneration122 topics
  5. Cytokines102 topics
  6. Microglia102 topics
  7. Anticancer Agents91 topic
  8. Heterocyclic Compounds91 topic
  9. Molecular Docking91 topic
  10. Virtual Screening91 topic
  11. Cisplatin81 topic
  12. Nephrotoxicity81 topic
  13. Amyloid Hypothesis71 topic
  14. Tau Pathology71 topic
  15. Microneedles61 topic
  16. Solid Lipid Nanoparticles61 topic
  17. Astrocytes51 topic
  18. Chitosan51 topic
  19. Depression51 topic
  20. Nanoparticles51 topic
  21. Synaptic Pruning51 topic
  22. Tryptophan Metabolism51 topic

Where does Ashif Iqubal work?

Ashif Iqubal's main affiliation in the publication record is Jamia Hamdard, India.

How many publications and citations does Ashif Iqubal have?

OpenAlex counts 86 works and 2,540 citations for Ashif Iqubal, an h-index of 26, ranking #571,578 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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