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Researcher · Universidade Federal do Rio Grande do Sul · Brazil

Tábita Hünemeier

Tábita Hünemeier publishes mostly in Genetics, Molecular Biology and Cell Biology, on topics such as Forensic and Genetic Research, melanin and skin pigmentation and Genetic Associations and Epidemiology.

ORCID 0000-0002-3156-2079 OpenAlex
World rank
#409,783
of 7,710,434 ranked researchers
Rank in Brazil
#1,884
of 253,246
Works
125
Citations
5.1k
Citations per work
41.2
h-index
32
i10-index 57Top 12% in Genetics (Biochemistry, Genetics and Molecular Biology)
Tábita Hünemeier

What does Tábita Hünemeier research?

Tábita Hünemeier's work is mostly in Biochemistry, Genetics and Molecular Biology (44%), Immunology and Microbiology (4%), Medicine (4%).

Life Sciences: 59 works · 47.4% of their workBiochemistry, Genetics and Molecular Biology: 55 works · 43.7% of their workGenetics: 37 works · 29.8% of their workMolecular Biology: 12 works · 9.6% of their workCell Biology: 5 works · 4.3% of their workImmunology and Microbiology: 5 works · 3.7% of their workImmunology: 5 works · 3.7% of their workEverything else: 57 works · 45.2% of their workHealth Sciences: 5 works · 3.7% of their workMedicine: 5 works · 3.7% of their workDermatology: 5 works · 3.7% of their workSocial Sciences: 5 works · 3.7% of their workArts and Humanities: 5 works · 3.7% of their workArcheology: 5 works · 3.7% of their workTHEIR WORKTábita Hünemeier125works
Life Sciences47%Health Sciences4%Social Sciences4%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology5543.7%
    2. Immunology and Microbiology53.7%
  2. Everything else5745.2%
    1. Medicine53.7%
    1. Arts and Humanities53.7%
  1. 1Genetics30% of their works
  2. 2Molecular Biology10% of their works
  3. 3Cell Biology4% of their works
  4. 4Immunology4% of their works
  5. 5Archeology4% of their works
  6. 6Dermatology4% of their works

Research topics

Which keywords describe Tábita Hünemeier's research?

The keywords of their largest research topics: Population Genetics, Ancient DNA, Genomic Analysis, Genetics, Genome-Wide Association, Quantitative Genetics, Genetic Diversity and Melanin.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Population Genetics384 topics
  2. Ancient DNA251 topic
  3. Genomic Analysis251 topic
  4. Genetics242 topics
  5. Genome-Wide Association192 topics
  6. Quantitative Genetics182 topics
  7. Genetic Diversity172 topics
  8. Melanin141 topic
  9. Pigmentation141 topic
  10. Genetic Variation111 topic
  11. Haplotype Mapping111 topic
  12. Biomedical Research101 topic
  13. Geometric Morphometrics101 topic
  14. Morphological Integration101 topic
  15. Race101 topic
  16. Genetic Structure91 topic
  17. Population Dynamics91 topic
  18. Genomic Selection81 topic
  19. Oxidative Stress81 topic
  20. Plant Breeding81 topic
  21. UV Radiation81 topic

Where does Tábita Hünemeier work?

Tábita Hünemeier's main affiliation in the publication record is Universidade Federal do Rio Grande do Sul, Brazil.

How many publications and citations does Tábita Hünemeier have?

OpenAlex counts 125 works and 5,148 citations for Tábita Hünemeier, an h-index of 32, ranking #409,783 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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