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Researcher · Animal Health Trust · United Kingdom

Matthew M Binns

Matthew M Binns publishes mostly in Genetics, Molecular Biology and Epidemiology, on topics such as Genetic and phenotypic traits in livestock, Animal Virus Infections Studies and Genetic Mapping and Diversity in Plants and Animals.

OpenAlex
World rank
#668,306
of 7,710,434 ranked researchers
Rank in United Kingdom
#56,566
of 367,258
Works
182
Citations
9.9k
Citations per work
54.5
h-index
53
i10-index 130Top 4.1% in Genetics (Biochemistry, Genetics and Molecular Biology)
Matthew M Binns

What does Matthew M Binns research?

Matthew M Binns's work is mostly in Biochemistry, Genetics and Molecular Biology (40%), Medicine (15%), Agricultural and Biological Sciences (11%).

Life Sciences: 93 works · 51.1% of their workBiochemistry, Genetics and Molecular Biology: 73 works · 39.9% of their workGenetics: 50 works · 27.7% of their workMolecular Biology: 22 works · 12.2% of their workAgricultural and Biological Sciences: 20 works · 11.2% of their workAnimal Science and Zoology: 11 works · 5.9% of their workPlant Science: 10 works · 5.3% of their workEverything else: 61 works · 33.5% of their workHealth Sciences: 28 works · 15.4% of their workMedicine: 28 works · 15.4% of their workEpidemiology: 18 works · 10.1% of their workInfectious Diseases: 10 works · 5.3% of their workTHEIR WORKMatthew M Binns182works
Life Sciences51%Health Sciences15%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology7339.9%
    2. Agricultural and Biological Sciences2011.2%
  2. Everything else6133.5%
    1. Medicine2815.4%
  1. 1Genetics28% of their works
  2. 2Molecular Biology12% of their works
  3. 3Epidemiology10% of their works
  4. 4Animal Science and Zoology6% of their works
  5. 5Plant Science5% of their works
  6. 6Infectious Diseases5% of their works

Research topics

Which keywords describe Matthew M Binns's research?

The keywords of their largest research topics: Epidemiology, Vaccine, Marker-Assisted Selection, Genetic Diversity, Population Genetics, Immune Response, Pathogenesis and Genomic Selection.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Epidemiology693 topics
  2. Vaccine693 topics
  3. Marker-Assisted Selection612 topics
  4. Genetic Diversity562 topics
  5. Population Genetics562 topics
  6. Immune Response492 topics
  7. Pathogenesis472 topics
  8. Genomic Selection361 topic
  9. Plant Breeding361 topic
  10. Coronavirus271 topic
  11. Porcine Reproductive and Respiratory Syndrome Virus271 topic
  12. Genome-wide Association Studies251 topic
  13. Quantitative Trait Loci251 topic
  14. Herpes Zoster221 topic
  15. Herpesviruses221 topic
  16. Genetic Structure201 topic
  17. Norovirus201 topic
  18. Population Dynamics201 topic
  19. Rotavirus201 topic
  20. Genetic Modification181 topic
  21. Transgenic Animals181 topic
  22. RNA-Seq161 topic
  23. sequence alignment161 topic

Where does Matthew M Binns work?

Matthew M Binns's main affiliation in the publication record is Animal Health Trust, United Kingdom.

How many publications and citations does Matthew M Binns have?

OpenAlex counts 182 works and 9,915 citations for Matthew M Binns, an h-index of 53, ranking #668,306 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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