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Researcher · Kyowa Kirin (Japan) · Japan

Mitsuo Satoh

Mitsuo Satoh publishes mostly in Molecular Biology, Radiology, Nuclear Medicine and Imaging and Materials Chemistry, on topics such as Monoclonal and Polyclonal Antibodies Research, Glycosylation and Glycoproteins Research and Solid-state spectroscopy and crystallography.

OpenAlex
World rank
#734,448
of 7,710,434 ranked researchers
Rank in Japan
#8,743
of 383,136
Works
120
Citations
6.5k
Citations per work
54.0
h-index
30
i10-index 54Top 15% in Molecular Biology
Mitsuo Satoh

What does Mitsuo Satoh research?

Mitsuo Satoh's work is mostly in Biochemistry, Genetics and Molecular Biology (22%), Medicine (11%), Materials Science (9%).

Everything else: 49 works · 40.9% of their workLife Sciences: 33 works · 27.6% of their workBiochemistry, Genetics and Molecular Biology: 26 works · 21.9% of their workMolecular Biology: 26 works · 21.9% of their workImmunology and Microbiology: 7 works · 5.7% of their workImmunology: 7 works · 5.7% of their workPhysical Sciences: 24 works · 20.4% of their workMaterials Science: 10 works · 8.7% of their workMaterials Chemistry: 10 works · 8.7% of their workChemistry: 9 works · 7.5% of their workSpectroscopy: 9 works · 7.5% of their workPhysics and Astronomy: 5 works · 4.2% of their workAtomic and Molecular Physics, and Optics: 5 works · 4.2% of their workHealth Sciences: 13 works · 11.1% of their workMedicine: 13 works · 11.1% of their workRadiology, Nuclear Medicine and Imaging: 13 works · 11.1% of their workTHEIR WORKMitsuo Satoh120works
Life Sciences28%Physical Sciences20%Health Sciences11%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else4940.9%
    1. Biochemistry, Genetics and Molecular Biology2621.9%
    2. Immunology and Microbiology75.7%
    1. Materials Science108.7%
    2. Chemistry97.5%
    3. Physics and Astronomy54.2%
    1. Medicine1311.1%
  1. 1Molecular Biology22% of their works
  2. 2Radiology, Nuclear Medicine and Imaging11% of their works
  3. 3Materials Chemistry9% of their works
  4. 4Spectroscopy8% of their works
  5. 5Immunology6% of their works
  6. 6Atomic and Molecular Physics, and Optics4% of their works

Research topics

Which keywords describe Mitsuo Satoh's research?

The keywords of their largest research topics: Glycosylation, Cancer, Fc Receptors, Therapeutic Antibodies, Glycans, Immune System, Protonic Conduction and Solid Acids.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Glycosylation803 topics
  2. Cancer362 topics
  3. Fc Receptors361 topic
  4. Therapeutic Antibodies361 topic
  5. Glycans291 topic
  6. Immune System291 topic
  7. Protonic Conduction241 topic
  8. Solid Acids241 topic
  9. NMR Spectroscopy201 topic
  10. Solid-State NMR201 topic
  11. Mammalian Cells151 topic
  12. Recombinant Protein151 topic
  13. Gene Therapy81 topic
  14. Viral Vectors81 topic
  15. Diffusion71 topic
  16. Galectins71 topic
  17. Immune Regulation71 topic
  18. Nuclear Magnetic Resonance71 topic

Where does Mitsuo Satoh work?

Mitsuo Satoh's main affiliation in the publication record is Kyowa Kirin (Japan), Japan.

How many publications and citations does Mitsuo Satoh have?

OpenAlex counts 120 works and 6,478 citations for Mitsuo Satoh, an h-index of 30, ranking #734,448 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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