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Researcher · University of Oslo · Norway

Ole A. Andreassen

Ole A. Andreassen publishes mostly in Genetics, Psychiatry and Mental health and Cognitive Neuroscience, on topics such as Genetic Associations and Epidemiology, Schizophrenia research and treatment and Functional Brain Connectivity Studies.

ORCID 0000-0002-4461-3568 OpenAlex
World rank
#16,028
of 7,710,434 ranked researchers
Rank in Norway
#52
of 33,005
Works
2,266
Citations
134k
Citations per work
59.3
h-index
157
i10-index 1,165Top 0.1% in Genetics (Biochemistry, Genetics and Molecular Biology)
Ole A. Andreassen

What does Ole A. Andreassen research?

Ole A. Andreassen's work is mostly in Biochemistry, Genetics and Molecular Biology (26%), Medicine (20%), Neuroscience (9%).

Everything else: 920 works · 40.6% of their workLife Sciences: 789 works · 34.8% of their workBiochemistry, Genetics and Molecular Biology: 582 works · 25.7% of their workGenetics: 417 works · 18.4% of their workMolecular Biology: 165 works · 7.3% of their workNeuroscience: 206 works · 9.1% of their workCognitive Neuroscience: 206 works · 9.1% of their workHealth Sciences: 444 works · 19.6% of their workMedicine: 444 works · 19.6% of their workPsychiatry and Mental health: 342 works · 15.1% of their workRadiology, Nuclear Medicine and Imaging: 102 works · 4.5% of their workSocial Sciences: 113 works · 5.0% of their workPsychology: 113 works · 5.0% of their workClinical Psychology: 113 works · 5.0% of their workTHEIR WORKOle A. Andreassen2,266works
Life Sciences35%Health Sciences20%Social Sciences5%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else92040.6%
    1. Biochemistry, Genetics and Molecular Biology58225.7%
    2. Neuroscience2069.1%
    1. Medicine44419.6%
    1. Psychology1135.0%
  1. 1Genetics18% of their works
  2. 2Psychiatry and Mental health15% of their works
  3. 3Cognitive Neuroscience9% of their works
  4. 4Molecular Biology7% of their works
  5. 5Clinical Psychology5% of their works
  6. 6Radiology, Nuclear Medicine and Imaging4% of their works

Research topics

Which keywords describe Ole A. Andreassen's research?

The keywords of their largest research topics: Genetic Variation, Genome-Wide Association, Neuroimaging, Antipsychotic Drugs, Neurocognitive Deficits, Brain Development, Functional Connectivity and Resting-State fMRI.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 18 words, with their numbers
  1. Genetic Variation6071 topic
  2. Genome-Wide Association6071 topic
  3. Neuroimaging4792 topics
  4. Antipsychotic Drugs4361 topic
  5. Neurocognitive Deficits4361 topic
  6. Brain Development3322 topics
  7. Functional Connectivity3081 topic
  8. Resting-State fMRI3081 topic
  9. Bipolar Spectrum Disorder2781 topic
  10. Prevalence2781 topic
  11. Diffusion MRI2011 topic
  12. White Matter2011 topic
  13. Cytokines1931 topic
  14. Neuroinflammation1931 topic
  15. Fragile X Syndrome1311 topic
  16. Rett Syndrome1311 topic
  17. Gene Set Enrichment Analysis991 topic
  18. Protein-Protein Interaction Networks991 topic

Where does Ole A. Andreassen work?

Ole A. Andreassen's main affiliation in the publication record is University of Oslo, Norway.

How many publications and citations does Ole A. Andreassen have?

OpenAlex counts 2,266 works and 134,385 citations for Ole A. Andreassen, an h-index of 157, ranking #16,028 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Other researchers named Ole A. Andreassen

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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