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Researcher · University of Amsterdam · Netherlands

Rob M. A. de Bie

Rob M. A. de Bie publishes mostly in Neurology, Cellular and Molecular Neuroscience and Neurology, on topics such as Neurological disorders and treatments, Parkinson's Disease Mechanisms and Treatments and Transcranial Magnetic Stimulation Studies.

ORCID 0000-0001-5267-0784 OpenAlex
World rank
#145,337
of 7,710,434 ranked researchers
Rank in Netherlands
#3,567
of 107,149
Works
256
Citations
14k
Citations per work
55.8
h-index
58
i10-index 138Top 2.1% in Neurology (Medicine)
Rob M. A. de Bie

What does Rob M. A. de Bie research?

Rob M. A. de Bie's work is mostly in Medicine (60%), Neuroscience (19%), Biochemistry, Genetics and Molecular Biology (2%).

Health Sciences: 154 works · 60.3% of their workMedicine: 154 works · 60.3% of their workNeurology: 148 works · 57.9% of their workPhysiology: 6 works · 2.4% of their workLife Sciences: 55 works · 21.3% of their workNeuroscience: 50 works · 19.4% of their workCellular and Molecular Neuroscience: 22 works · 8.4% of their workNeurology: 20 works · 7.7% of their workCognitive Neuroscience: 8 works · 3.3% of their workBiochemistry, Genetics and Molecular Biology: 5 works · 1.9% of their workMolecular Biology: 5 works · 1.9% of their workEverything else: 47 works · 18.4% of their workTHEIR WORKRob M. A. de Bie256works
Life Sciences21%Health Sciences60%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine15460.3%
    1. Neuroscience5019.4%
    2. Biochemistry, Genetics and Molecular Biology51.9%
  3. Everything else4718.4%
  1. 1Neurology58% of their works
  2. 2Cellular and Molecular Neuroscience8% of their works
  3. 3Neurology8% of their works
  4. 4Cognitive Neuroscience3% of their works
  5. 5Physiology2% of their works
  6. 6Molecular Biology2% of their works

Research topics

Which keywords describe Rob M. A. de Bie's research?

The keywords of their largest research topics: Parkinson's Disease, Neurological Disorders, Deep Brain Stimulation, Epilepsy, Mitochondrial Dysfunction, Basal Ganglia, Subthalamic Nucleus and Neurodegeneration.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Parkinson's Disease3773 topics
  2. Neurological Disorders2473 topics
  3. Deep Brain Stimulation2132 topics
  4. Epilepsy1982 topics
  5. Mitochondrial Dysfunction1882 topics
  6. Basal Ganglia1851 topic
  7. Subthalamic Nucleus1851 topic
  8. Neurodegeneration1772 topics
  9. Dopaminergic Neurons1641 topic
  10. a-Synuclein1641 topic
  11. Transcranial Direct Current Stimulation481 topic
  12. Transcranial Magnetic Stimulation481 topic
  13. Botulinum Toxin341 topic
  14. Neurotoxins341 topic
  15. Camptocormia281 topic
  16. Dropped Head Syndrome281 topic
  17. Huntington's Disease241 topic
  18. Protein Aggregation241 topic
  19. Electrode Arrays171 topic
  20. Neural Stimulation171 topic
  21. Glycogen131 topic
  22. Metabolism131 topic

Where does Rob M. A. de Bie work?

Rob M. A. de Bie's main affiliation in the publication record is University of Amsterdam, Netherlands.

How many publications and citations does Rob M. A. de Bie have?

OpenAlex counts 256 works and 14,294 citations for Rob M. A. de Bie, an h-index of 58, ranking #145,337 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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