Science Explorer Back to mapMap
Discussion
Researcher · Furman University · United States

Adi D. Dubash

Adi D. Dubash publishes mostly in Molecular Biology, Cell Biology and Cardiology and Cardiovascular Medicine, on topics such as Wnt/β-catenin signaling in development and cancer, Protein Kinase Regulation and GTPase Signaling and Cellular Mechanics and Interactions.

ORCID 0000-0001-5595-5355 OpenAlex
World rank
#1,506,478
of 7,710,434 ranked researchers
Rank in United States
#471,957
of 1,627,098
Works
48
Citations
1.6k
Citations per work
34.1
h-index
18
i10-index 19Top 39% in Molecular Biology
Adi D. Dubash

What does Adi D. Dubash research?

Adi D. Dubash's work is mostly in Biochemistry, Genetics and Molecular Biology (60%), Medicine (22%).

Life Sciences: 29 works · 60.5% of their workBiochemistry, Genetics and Molecular Biology: 29 works · 60.5% of their workMolecular Biology: 21 works · 44.0% of their workCell Biology: 8 works · 16.5% of their workHealth Sciences: 11 works · 22.1% of their workMedicine: 11 works · 22.1% of their workCardiology and Cardiovascular Medicine: 7 works · 13.8% of their workImmunology and Allergy: 2 works · 3.7% of their workOncology: 1 works · 2.8% of their workDermatology: 1 works · 1.8% of their workEverything else: 8 works · 17.4% of their workTHEIR WORKAdi D. Dubash48works
Life Sciences60%Health Sciences22%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology2960.5%
    1. Medicine1122.1%
  3. Everything else817.4%
  1. 1Molecular Biology44% of their works
  2. 2Cell Biology16% of their works
  3. 3Cardiology and Cardiovascular Medicine14% of their works
  4. 4Immunology and Allergy4% of their works
  5. 5Oncology3% of their works
  6. 6Dermatology2% of their works

Research topics

Which keywords describe Adi D. Dubash's research?

The keywords of their largest research topics: Stem Cells, Cancer, Cell Migration, Wnt Signaling, ß-Catenin, Extracellular Matrix, Mechanotransduction and Protein Kinases.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Stem Cells253 topics
  2. Cancer192 topics
  3. Cell Migration162 topics
  4. Wnt Signaling151 topic
  5. ß-Catenin151 topic
  6. Extracellular Matrix102 topics
  7. Mechanotransduction102 topics
  8. Protein Kinases101 topic
  9. Ras Signaling101 topic
  10. Cardiac Remodeling92 topics
  11. Genetic Mutations92 topics
  12. Cell Mechanics61 topic
  13. Cytoskeleton61 topic
  14. Arrhythmogenic Right Ventricular Cardiomyopathy51 topic
  15. Intermediate Filaments51 topic
  16. Keratins51 topic
  17. Sudden Death51 topic
  18. Cardiomyopathies41 topic
  19. Hippo Pathway41 topic
  20. Inflammatory Response41 topic
  21. Myocardial Infarction41 topic
  22. Myosin41 topic
  23. YAP/TAZ41 topic

Where does Adi D. Dubash work?

Adi D. Dubash's main affiliation in the publication record is Furman University, United States.

How many publications and citations does Adi D. Dubash have?

OpenAlex counts 48 works and 1,638 citations for Adi D. Dubash, an h-index of 18, ranking #1,506,478 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Adi D. Dubash. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.