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Researcher · University of Tübingen · Germany

Albrecht Lepple‐Wienhues

Albrecht Lepple‐Wienhues publishes mostly in Molecular Biology, Cellular and Molecular Neuroscience and Physiology, on topics such as Ion channel regulation and function, Neuroscience and Neuropharmacology Research and Neuroscience and Neural Engineering.

OpenAlex
World rank
#1,493,847
of 7,710,434 ranked researchers
Rank in Germany
#73,649
of 364,343
Works
34
Citations
1.7k
Citations per work
49.4
h-index
17
i10-index 18Top 42% in Molecular Biology
Albrecht Lepple‐Wienhues

What does Albrecht Lepple‐Wienhues research?

Albrecht Lepple‐Wienhues's work is mostly in Biochemistry, Genetics and Molecular Biology (30%), Neuroscience (15%), Medicine (12%).

Life Sciences: 17 works · 50.0% of their workBiochemistry, Genetics and Molecular Biology: 10 works · 30.2% of their workMolecular Biology: 10 works · 30.2% of their workNeuroscience: 5 works · 15.1% of their workCellular and Molecular Neuroscience: 5 works · 15.1% of their workImmunology and Microbiology: 2 works · 4.7% of their workImmunology: 2 works · 4.7% of their workEverything else: 11 works · 32.5% of their workHealth Sciences: 4 works · 11.7% of their workMedicine: 4 works · 11.7% of their workPhysiology: 2 works · 7.0% of their workOncology: 2 works · 4.7% of their workPhysical Sciences: 2 works · 5.8% of their workEngineering: 2 works · 5.8% of their workBiomedical Engineering: 2 works · 5.8% of their workTHEIR WORKAlbrechtLepple‐Wienhues34works
Life Sciences50%Physical Sciences6%Health Sciences12%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology1030.2%
    2. Neuroscience515.1%
    3. Immunology and Microbiology24.7%
  2. Everything else1132.5%
    1. Medicine411.7%
    1. Engineering25.8%
  1. 1Molecular Biology30% of their works
  2. 2Cellular and Molecular Neuroscience15% of their works
  3. 3Physiology7% of their works
  4. 4Biomedical Engineering6% of their works
  5. 5Oncology5% of their works
  6. 6Immunology5% of their works

Research topics

Which keywords describe Albrecht Lepple‐Wienhues's research?

The keywords of their largest research topics: Ion Channels, Calcium Channels, Voltage-Gated Channels, Electrode Arrays, Neural Stimulation, Neurotransmission, Synaptic Plasticity and Apoptosis.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 17 words, with their numbers
  1. Ion Channels132 topics
  2. Calcium Channels101 topic
  3. Voltage-Gated Channels101 topic
  4. Electrode Arrays51 topic
  5. Neural Stimulation51 topic
  6. Neurotransmission51 topic
  7. Synaptic Plasticity51 topic
  8. Apoptosis41 topic
  9. Cell Death41 topic
  10. Calcium Signaling31 topic
  11. Channelrhodopsin31 topic
  12. Ion-Selective Electrodes31 topic
  13. Lab-on-a-Chip31 topic
  14. Optical Sensors31 topic
  15. Optogenetics31 topic
  16. Poly(dimethylsiloxane)31 topic
  17. TRP Channels31 topic

Where does Albrecht Lepple‐Wienhues work?

Albrecht Lepple‐Wienhues's main affiliation in the publication record is University of Tübingen, Germany.

How many publications and citations does Albrecht Lepple‐Wienhues have?

OpenAlex counts 34 works and 1,679 citations for Albrecht Lepple‐Wienhues, an h-index of 17, ranking #1,493,847 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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