David M. Holtzman
David M. Holtzman publishes mostly in Physiology, Molecular Biology and Psychiatry and Mental health, on topics such as Alzheimer's disease research and treatments, Dementia and Cognitive Impairment Research and Neuroinflammation and Neurodegeneration Mechanisms.
- World rank
- #1,030 of 7,710,434 ranked researchers
- Rank in United States
- #434 of 1,627,098
- Works
- 1,037
- Citations
- 176k
- Citations per work
- 169.4
What does David M. Holtzman research?
David M. Holtzman's work is mostly in Medicine (34%), Neuroscience (19%), Biochemistry, Genetics and Molecular Biology (12%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Everything else35834.5%
- Health Sciences35734.4%
- Medicine35734.4%
- Life Sciences32331.1%
- Neuroscience19618.9%
- Biochemistry, Genetics and Molecular Biology12712.2%
- 1Physiology24% of their works
- 2Molecular Biology12% of their works
- 3Psychiatry and Mental health11% of their works
- 4Neurology9% of their works
- 5Cellular and Molecular Neuroscience6% of their works
- 6Cognitive Neuroscience4% of their works
Research topics
- Alzheimer's disease research and treatments
- Dementia and Cognitive Impairment Research
- Neuroinflammation and Neurodegeneration Mechanisms
- Cholinesterase and Neurodegenerative Diseases
- Neuroscience and Neuropharmacology Research
- Neurological Disease Mechanisms and Treatments
- Functional Brain Connectivity Studies
- Computational Drug Discovery Methods
Which keywords describe David M. Holtzman's research?
The keywords of their largest research topics: Neurodegeneration, Neuroinflammation, Oxidative Stress, Amyloid Hypothesis, Tau Pathology, Alzheimer's Disease, Dementia and Mild Cognitive Impairment.
- Virtual Screening
- Resting-State fMRI
- Vascular Cognitive Impairment
- Synaptic Plasticity
- Cholinesterase Inhibitors
- Neurological Disorders
- Microglia
- Mild Cognitive Impairment
- Alzheimer's Disease
- Amyloid Hypothesis
- Neuroinflammation
- Neurodegeneration
- Oxidative Stress
- Tau Pathology
- Dementia
- Astrocytes
- Neurodegenerative Diseases
- Acetylcholinesterase
- Neurotransmission
- Cerebral Hypoperfusion
- Functional Connectivity
- Molecular Docking
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.
All 22 words, with their numbers
- Neurodegeneration7522 topics
- Neuroinflammation7522 topics
- Oxidative Stress6412 topics
- Amyloid Hypothesis5901 topic
- Tau Pathology5901 topic
- Alzheimer's Disease3462 topics
- Dementia2841 topic
- Mild Cognitive Impairment2841 topic
- Astrocytes1621 topic
- Microglia1621 topic
- Neurodegenerative Diseases1132 topics
- Neurological Disorders1002 topics
- Acetylcholinesterase621 topic
- Cholinesterase Inhibitors621 topic
- Neurotransmission531 topic
- Synaptic Plasticity531 topic
- Cerebral Hypoperfusion511 topic
- Vascular Cognitive Impairment511 topic
- Functional Connectivity471 topic
- Resting-State fMRI471 topic
- Molecular Docking461 topic
- Virtual Screening461 topic
Where does David M. Holtzman work?
David M. Holtzman's main affiliation in the publication record is Washington University in St. Louis, United States.
How many publications and citations does David M. Holtzman have?
OpenAlex counts 1,037 works and 175,684 citations for David M. Holtzman, an h-index of 208, ranking #1,030 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
Discussion
Ask a question or share a note about David M. Holtzman. Posts are plain text and can be reported.