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Researcher · Eppendorf (Germany) · Germany

Hasso Scholz

Hasso Scholz publishes mostly in Cardiology and Cardiovascular Medicine, Molecular Biology and Physiology, on topics such as Cardiac electrophysiology and arrhythmias, Ion channel regulation and function and Receptor Mechanisms and Signaling.

OpenAlex
World rank
#1,212,169
of 7,710,434 ranked researchers
Rank in Germany
#59,687
of 364,343
Works
298
Citations
11k
Citations per work
36.0
h-index
52
i10-index 157Top 2.8% in Cardiology and Cardiovascular Medicine
Hasso Scholz

What does Hasso Scholz research?

Hasso Scholz's work is mostly in Medicine (41%), Biochemistry, Genetics and Molecular Biology (24%), Neuroscience (3%).

Health Sciences: 123 works · 41.4% of their workMedicine: 123 works · 41.4% of their workCardiology and Cardiovascular Medicine: 103 works · 34.7% of their workPhysiology: 13 works · 4.4% of their workSurgery: 7 works · 2.3% of their workEverything else: 85 works · 28.6% of their workLife Sciences: 83 works · 27.7% of their workBiochemistry, Genetics and Molecular Biology: 72 works · 24.3% of their workMolecular Biology: 72 works · 24.3% of their workNeuroscience: 10 works · 3.4% of their workCellular and Molecular Neuroscience: 10 works · 3.4% of their workPhysical Sciences: 7 works · 2.3% of their workChemistry: 7 works · 2.3% of their workAnalytical Chemistry: 7 works · 2.3% of their workTHEIR WORKHasso Scholz298works
Life Sciences28%Physical Sciences2%Health Sciences41%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine12341.4%
  2. Everything else8528.6%
    1. Biochemistry, Genetics and Molecular Biology7224.3%
    2. Neuroscience103.4%
    1. Chemistry72.3%
  1. 1Cardiology and Cardiovascular Medicine35% of their works
  2. 2Molecular Biology24% of their works
  3. 3Physiology4% of their works
  4. 4Cellular and Molecular Neuroscience3% of their works
  5. 5Surgery2% of their works
  6. 6Analytical Chemistry2% of their works

Research topics

Which keywords describe Hasso Scholz's research?

The keywords of their largest research topics: Ion Channels, Sudden Cardiac Death, Cell Signaling, Arrhythmias, Long-QT Syndrome, Calcium Channels, Voltage-Gated Channels and Heart Failure.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Ion Channels1942 topics
  2. Sudden Cardiac Death1322 topics
  3. Cell Signaling1222 topics
  4. Arrhythmias1131 topic
  5. Long-QT Syndrome1131 topic
  6. Calcium Channels811 topic
  7. Voltage-Gated Channels811 topic
  8. Heart Failure663 topics
  9. Crystal Structure411 topic
  10. G-Protein-Coupled Receptors411 topic
  11. Echocardiography392 topics
  12. Cardiomyopathies271 topic
  13. Diagnosis271 topic
  14. Genetic Mutations271 topic
  15. Guidelines271 topic
  16. Endothelial Dysfunction241 topic
  17. Nitric Oxide241 topic
  18. Chamber Quantification201 topic
  19. Diastolic Function201 topic
  20. Cardiac Resynchronization Therapy191 topic
  21. Implantable Cardioverter-Defibrillator191 topic

Where does Hasso Scholz work?

Hasso Scholz's main affiliation in the publication record is Eppendorf (Germany), Germany.

How many publications and citations does Hasso Scholz have?

OpenAlex counts 298 works and 10,736 citations for Hasso Scholz, an h-index of 52, ranking #1,212,169 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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