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Researcher · University of Nebraska Medical Center · United States

Howard Eliot Gendelman

Howard Eliot Gendelman publishes mostly in Virology, Neurology and Infectious Diseases, on topics such as HIV Research and Treatment, Neuroinflammation and Neurodegeneration Mechanisms and Alzheimer's disease research and treatments.

ORCID 0000-0002-7831-0370 OpenAlex
World rank
#49,967
of 7,710,434 ranked researchers
Rank in United States
#19,287
of 1,627,098
Works
655
Citations
48k
Citations per work
73.3
h-index
111
i10-index 446Top 0.5% in Virology
Howard Eliot Gendelman

What does Howard Eliot Gendelman research?

Howard Eliot Gendelman's work is mostly in Immunology and Microbiology (24%), Medicine (16%), Neuroscience (12%).

Life Sciences: 288 works · 44.0% of their workImmunology and Microbiology: 160 works · 24.4% of their workVirology: 119 works · 18.1% of their workImmunology: 41 works · 6.3% of their workNeuroscience: 77 works · 11.8% of their workNeurology: 77 works · 11.8% of their workBiochemistry, Genetics and Molecular Biology: 51 works · 7.8% of their workMolecular Biology: 51 works · 7.8% of their workEverything else: 265 works · 40.5% of their workHealth Sciences: 102 works · 15.5% of their workMedicine: 102 works · 15.5% of their workInfectious Diseases: 69 works · 10.5% of their workPhysiology: 33 works · 5.0% of their workTHEIR WORKHoward EliotGendelman655works
Life Sciences44%Health Sciences16%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Immunology and Microbiology16024.4%
    2. Neuroscience7711.8%
    3. Biochemistry, Genetics and Molecular Biology517.8%
  2. Everything else26540.5%
    1. Medicine10215.5%
  1. 1Virology18% of their works
  2. 2Neurology12% of their works
  3. 3Infectious Diseases10% of their works
  4. 4Molecular Biology8% of their works
  5. 5Immunology6% of their works
  6. 6Physiology5% of their works

Research topics

Which keywords describe Howard Eliot Gendelman's research?

The keywords of their largest research topics: Antiretroviral Therapy, Immune Activation, Neuroinflammation, Neurodegeneration, AIDS, HIV, Cytokines and Microglia.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 24 words, with their numbers
  1. Antiretroviral Therapy5794 topics
  2. Immune Activation3822 topics
  3. Neuroinflammation3453 topics
  4. Neurodegeneration3423 topics
  5. AIDS3341 topic
  6. HIV3341 topic
  7. Cytokines2552 topics
  8. Microglia2552 topics
  9. Astrocytes2071 topic
  10. Synaptic Pruning2071 topic
  11. Oxidative Stress1833 topics
  12. HIV Infection1662 topics
  13. Amyloid Hypothesis901 topic
  14. Tau Pathology901 topic
  15. Cardiovascular Disease851 topic
  16. Inflammation851 topic
  17. Drug Resistance811 topic
  18. Integrase Inhibitors811 topic
  19. HIV Transmission791 topic
  20. Preexposure Prophylaxis791 topic
  21. Depression481 topic
  22. Tryptophan Metabolism481 topic
  23. Parkinson's Disease451 topic
  24. a-Synuclein451 topic

Where does Howard Eliot Gendelman work?

Howard Eliot Gendelman's main affiliation in the publication record is University of Nebraska Medical Center, United States.

How many publications and citations does Howard Eliot Gendelman have?

OpenAlex counts 655 works and 47,995 citations for Howard Eliot Gendelman, an h-index of 111, ranking #49,967 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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