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Researcher · Universidade Federal Fluminense · Brazil

João Paulo Lima Daher

João Paulo Lima Daher publishes mostly in Neurology, Physiology and Molecular Biology, on topics such as Parkinson's Disease Mechanisms and Treatments, Neurological diseases and metabolism and Alzheimer's disease research and treatments.

ORCID 0009-0006-6783-4466 OpenAlex
World rank
#809,661
of 7,710,434 ranked researchers
Rank in Brazil
#4,707
of 253,246
Works
32
Citations
2.1k
Citations per work
65.8
h-index
17
i10-index 20Top 31% in Neurology (Medicine)
João Paulo Lima Daher

What does João Paulo Lima Daher research?

João Paulo Lima Daher's work is mostly in Medicine (36%), Neuroscience (13%), Biochemistry, Genetics and Molecular Biology (8%).

Everything else: 14 works · 43.8% of their workHealth Sciences: 11 works · 35.6% of their workMedicine: 11 works · 35.6% of their workNeurology: 7 works · 20.7% of their workPhysiology: 3 works · 8.0% of their workCardiology and Cardiovascular Medicine: 2 works · 6.9% of their workLife Sciences: 7 works · 20.6% of their workNeuroscience: 4 works · 12.6% of their workNeurology: 2 works · 6.9% of their workCellular and Molecular Neuroscience: 2 works · 5.7% of their workBiochemistry, Genetics and Molecular Biology: 3 works · 8.0% of their workMolecular Biology: 3 works · 8.0% of their workTHEIR WORKJoão Paulo LimaDaher32works
Life Sciences21%Health Sciences36%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else1443.8%
    1. Medicine1135.6%
    1. Neuroscience412.6%
    2. Biochemistry, Genetics and Molecular Biology38.0%
  1. 1Neurology21% of their works
  2. 2Physiology8% of their works
  3. 3Molecular Biology8% of their works
  4. 4Cardiology and Cardiovascular Medicine7% of their works
  5. 5Neurology7% of their works
  6. 6Cellular and Molecular Neuroscience6% of their works

Research topics

Which keywords describe João Paulo Lima Daher's research?

The keywords of their largest research topics: Neurodegeneration, Parkinson's Disease, Mitochondrial Dysfunction, Oxidative Stress, Dopaminergic Neurons, a-Synuclein, Genetic Risk Factors and Amyloid Hypothesis.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Neurodegeneration244 topics
  2. Parkinson's Disease173 topics
  3. Mitochondrial Dysfunction162 topics
  4. Oxidative Stress162 topics
  5. Dopaminergic Neurons142 topics
  6. Genetic Risk Factors111 topic
  7. a-Synuclein111 topic
  8. Amyloid Hypothesis51 topic
  9. Brain Iron Accumulation51 topic
  10. Pantothenate Kinase51 topic
  11. Tau Pathology51 topic
  12. Ion Channels42 topics
  13. Deep Brain Stimulation31 topic
  14. NR4A231 topic
  15. Nurr131 topic
  16. Subthalamic Nucleus31 topic
  17. Antiretroviral Therapy21 topic
  18. Calcium Channels21 topic
  19. Calcium Signaling21 topic
  20. HIV Transmission21 topic
  21. TRP Channels21 topic
  22. Voltage-Gated Channels21 topic

Where does João Paulo Lima Daher work?

João Paulo Lima Daher's main affiliation in the publication record is Universidade Federal Fluminense, Brazil.

How many publications and citations does João Paulo Lima Daher have?

OpenAlex counts 32 works and 2,106 citations for João Paulo Lima Daher, an h-index of 17, ranking #809,661 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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