Katharina Maria Main
Katharina Maria Main publishes mostly in Endocrinology, Diabetes and Metabolism, Health, Toxicology and Mutagenesis and Reproductive Medicine, on topics such as Sexual Differentiation and Disorders, Effects and risks of endocrine disrupting chemicals and Growth Hormone and Insulin-like Growth Factors.
- World rank
- #82,062 of 7,710,434 ranked researchers
- Rank in Denmark
- #476 of 43,819
- Works
- 425
- Citations
- 26k
- Citations per work
- 60.4
What does Katharina Maria Main research?
Katharina Maria Main's work is mostly in Medicine (40%), Environmental Science (12%), Biochemistry, Genetics and Molecular Biology (9%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Health Sciences16939.8%
- Medicine16939.8%
- Everything else16538.9%
- Physical Sciences5111.9%
- Environmental Science5111.9%
- Life Sciences409.4%
- 1Endocrinology, Diabetes and Metabolism13% of their works
- 2Health, Toxicology and Mutagenesis12% of their works
- 3Reproductive Medicine12% of their works
- 4Pediatrics, Perinatology and Child Health10% of their works
- 5Molecular Biology9% of their works
- 6Public Health, Environmental and Occupational Health5% of their works
Research topics
- Sexual Differentiation and Disorders
- Effects and risks of endocrine disrupting chemicals
- Growth Hormone and Insulin-like Growth Factors
- Ovarian function and disorders
- Birth, Development, and Health
- Hypothalamic control of reproductive hormones
- Testicular diseases and treatments
- Toxic Organic Pollutants Impact
Which keywords describe Katharina Maria Main's research?
The keywords of their largest research topics: Environmental Exposure, Neuroendocrine Control, Metabolic Syndrome, Congenital Adrenal Hyperplasia, Steroid 21-Hydroxylase Deficiency, Bisphenol A, Endocrine-Disrupting Chemicals and IGF-1.
- Brominated Flame Retardants
- Germ Cell Tumors
- GPR54 Gene
- Early-Life Conditions
- Ovarian Reserve
- IGF-1
- Bisphenol A
- Congenital Adrenal Hyperplasia
- Neuroendocrine Control
- Environmental Exposure
- Metabolic Syndrome
- Steroid 21-Hydroxylase Deficiency
- Endocrine-Disrupting Chemicals
- Insulin-like Growth Factors
- Polycystic Ovary Syndrome
- Fetal Programming
- Puberty
- Testicular Cancer
- Polycyclic Aromatic Hydrocarbons
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 19 words, with their numbers
- Environmental Exposure1112 topics
- Neuroendocrine Control992 topics
- Metabolic Syndrome972 topics
- Congenital Adrenal Hyperplasia881 topic
- Steroid 21-Hydroxylase Deficiency881 topic
- Bisphenol A761 topic
- Endocrine-Disrupting Chemicals761 topic
- IGF-1571 topic
- Insulin-like Growth Factors571 topic
- Ovarian Reserve491 topic
- Polycystic Ovary Syndrome491 topic
- Early-Life Conditions481 topic
- Fetal Programming481 topic
- GPR54 Gene421 topic
- Puberty421 topic
- Germ Cell Tumors371 topic
- Testicular Cancer371 topic
- Brominated Flame Retardants351 topic
- Polycyclic Aromatic Hydrocarbons351 topic
Where does Katharina Maria Main work?
Katharina Maria Main's main affiliation in the publication record is Rigshospitalet, Denmark.
How many publications and citations does Katharina Maria Main have?
OpenAlex counts 425 works and 25,656 citations for Katharina Maria Main, an h-index of 87, ranking #82,062 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
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