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Researcher · Endokrinologikum · Germany

Klaus‐Peter Liesenkötter

Klaus‐Peter Liesenkötter publishes mostly in Endocrinology, Diabetes and Metabolism, Molecular Biology and Pediatrics, Perinatology and Child Health, on topics such as Thyroid Disorders and Treatments, Sexual Differentiation and Disorders and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities.

OpenAlex
World rank
#4,674,133
of 7,710,434 ranked researchers
Rank in Germany
#238,600
of 364,343
Works
18
Citations
331
Citations per work
18.4
h-index
9
i10-index 9Top 63% in Endocrinology, Diabetes and Metabolism
Klaus‐Peter Liesenkötter

What does Klaus‐Peter Liesenkötter research?

Klaus‐Peter Liesenkötter's work is mostly in Medicine (55%), Biochemistry, Genetics and Molecular Biology (15%).

Health Sciences: 10 works · 54.8% of their workMedicine: 10 works · 54.8% of their workEndocrinology, Diabetes and Metabolism: 6 works · 34.0% of their workPediatrics, Perinatology and Child Health: 2 works · 9.4% of their workPublic Health, Environmental and Occupational Health: 1 works · 5.7% of their workSurgery: 1 works · 5.7% of their workEverything else: 5 works · 30.1% of their workLife Sciences: 3 works · 15.1% of their workBiochemistry, Genetics and Molecular Biology: 3 works · 15.1% of their workMolecular Biology: 2 works · 9.4% of their workGenetics: 1 works · 5.7% of their workTHEIR WORKKlaus‐PeterLiesenkötter18works
Life Sciences15%Health Sciences55%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine1054.8%
  2. Everything else530.1%
    1. Biochemistry, Genetics and Molecular Biology315.1%
  1. 1Endocrinology, Diabetes and Metabolism34% of their works
  2. 2Molecular Biology9% of their works
  3. 3Pediatrics, Perinatology and Child Health9% of their works
  4. 4Genetics6% of their works
  5. 5Public Health, Environmental and Occupational Health6% of their works
  6. 6Surgery6% of their works

Research topics

Which keywords describe Klaus‐Peter Liesenkötter's research?

The keywords of their largest research topics: Genetic Variants, Thyroid Dysfunction, Thyroid Hormone, Cardiovascular Disease, Congenital Adrenal Hyperplasia, Diabetes, Steroid 21-Hydroxylase Deficiency and Obesity.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 20 words, with their numbers
  1. Genetic Variants82 topics
  2. Thyroid Dysfunction81 topic
  3. Thyroid Hormone81 topic
  4. Cardiovascular Disease52 topics
  5. Congenital Adrenal Hyperplasia51 topic
  6. Diabetes52 topics
  7. Steroid 21-Hydroxylase Deficiency51 topic
  8. Obesity42 topics
  9. Glucose-6-Phosphate Dehydrogenase Deficiency31 topic
  10. Hypoglycemia31 topic
  11. Insulin Therapy31 topic
  12. Neonatal Jaundice31 topic
  13. Sex Determination31 topic
  14. X Chromosome Inactivation31 topic
  15. Childhood21 topic
  16. Differentiated Thyroid Cancer21 topic
  17. Insulin Resistance21 topic
  18. Metabolic Syndrome21 topic
  19. Overweight21 topic
  20. Thyroid Nodules21 topic

Where does Klaus‐Peter Liesenkötter work?

Klaus‐Peter Liesenkötter's main affiliation in the publication record is Endokrinologikum, Germany.

How many publications and citations does Klaus‐Peter Liesenkötter have?

OpenAlex counts 18 works and 331 citations for Klaus‐Peter Liesenkötter, an h-index of 9, ranking #4,674,133 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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