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Researcher · Poznan University of Medical Sciences · Poland

Krzysztof Katulski

Krzysztof Katulski publishes mostly in Reproductive Medicine, Public Health, Environmental and Occupational Health and Endocrinology, Diabetes and Metabolism, on topics such as Ovarian function and disorders, Reproductive Biology and Fertility and Hypothalamic control of reproductive hormones.

ORCID 0000-0001-6206-3785 OpenAlex
World rank
#2,011,510
of 7,710,434 ranked researchers
Rank in Poland
#12,055
of 100,960
Works
35
Citations
999
Citations per work
28.5
h-index
13
i10-index 15Top 44% in Reproductive Medicine
Krzysztof Katulski

What does Krzysztof Katulski research?

Krzysztof Katulski's work is mostly in Medicine (57%), Biochemistry, Genetics and Molecular Biology (7%), Psychology (3%).

Health Sciences: 20 works · 57.3% of their workMedicine: 20 works · 57.3% of their workReproductive Medicine: 9 works · 25.8% of their workPublic Health, Environmental and Occupational Health: 5 works · 14.6% of their workEndocrinology, Diabetes and Metabolism: 4 works · 12.4% of their workObstetrics and Gynecology: 2 works · 4.5% of their workEverything else: 11 works · 32.6% of their workLife Sciences: 2 works · 6.7% of their workBiochemistry, Genetics and Molecular Biology: 2 works · 6.7% of their workGenetics: 2 works · 6.7% of their workSocial Sciences: 1 works · 3.4% of their workPsychology: 1 works · 3.4% of their workClinical Psychology: 1 works · 3.4% of their workTHEIR WORKKrzysztofKatulski35works
Life Sciences7%Health Sciences57%Social Sciences3%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine2057.3%
  2. Everything else1132.6%
    1. Biochemistry, Genetics and Molecular Biology26.7%
    1. Psychology13.4%
  1. 1Reproductive Medicine26% of their works
  2. 2Public Health, Environmental and Occupational Health15% of their works
  3. 3Endocrinology, Diabetes and Metabolism12% of their works
  4. 4Genetics7% of their works
  5. 5Obstetrics and Gynecology4% of their works
  6. 6Clinical Psychology3% of their works

Research topics

Which keywords describe Krzysztof Katulski's research?

The keywords of their largest research topics: Metabolic Syndrome, Obesity, Ovarian Reserve, Polycystic Ovary Syndrome, Neuroendocrine Control, Fertility Preservation, Ovarian Tissue Cryopreservation and Hormonal Regulation.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 20 words, with their numbers
  1. Metabolic Syndrome172 topics
  2. Obesity162 topics
  3. Ovarian Reserve141 topic
  4. Polycystic Ovary Syndrome141 topic
  5. Neuroendocrine Control112 topics
  6. Fertility Preservation91 topic
  7. Ovarian Tissue Cryopreservation91 topic
  8. Hormonal Regulation82 topics
  9. GPR54 Gene61 topic
  10. Puberty61 topic
  11. IGF-151 topic
  12. Insulin-like Growth Factors51 topic
  13. Hormone Therapy31 topic
  14. Sex Determination31 topic
  15. Testosterone31 topic
  16. X Chromosome Inactivation31 topic
  17. Congenital Adrenal Hyperplasia21 topic
  18. Ghrelin21 topic
  19. Leptin21 topic
  20. Steroid 21-Hydroxylase Deficiency21 topic

Where does Krzysztof Katulski work?

Krzysztof Katulski's main affiliation in the publication record is Poznan University of Medical Sciences, Poland.

How many publications and citations does Krzysztof Katulski have?

OpenAlex counts 35 works and 999 citations for Krzysztof Katulski, an h-index of 13, ranking #2,011,510 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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