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Researcher · 23andMe (United States) · United States

Matthew H. McIntyre

Matthew H. McIntyre publishes mostly in Genetics, Molecular Biology and Experimental and Cognitive Psychology, on topics such as Genetic Associations and Epidemiology, Parkinson's Disease Mechanisms and Treatments and Sexual Differentiation and Disorders.

ORCID 0000-0001-5791-6245 OpenAlex
World rank
#14,507
of 7,710,434 ranked researchers
Rank in United States
#5,117
of 1,627,098
Works
139
Citations
14k
Citations per work
100.7
h-index
51
i10-index 95Top 4.5% in Genetics (Biochemistry, Genetics and Molecular Biology)
Matthew H. McIntyre

What does Matthew H. McIntyre research?

Matthew H. McIntyre's work is mostly in Biochemistry, Genetics and Molecular Biology (31%), Medicine (7%), Psychology (6%).

Everything else: 73 works · 52.5% of their workLife Sciences: 48 works · 34.6% of their workBiochemistry, Genetics and Molecular Biology: 44 works · 31.3% of their workGenetics: 31 works · 22.0% of their workMolecular Biology: 13 works · 9.3% of their workNeuroscience: 5 works · 3.3% of their workCognitive Neuroscience: 5 works · 3.3% of their workHealth Sciences: 10 works · 7.4% of their workMedicine: 10 works · 7.4% of their workNeurology: 6 works · 4.1% of their workEpidemiology: 5 works · 3.3% of their workSocial Sciences: 8 works · 5.5% of their workPsychology: 8 works · 5.5% of their workExperimental and Cognitive Psychology: 8 works · 5.5% of their workTHEIR WORKMatthew H.McIntyre139works
Life Sciences35%Health Sciences7%Social Sciences6%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else7352.5%
    1. Biochemistry, Genetics and Molecular Biology4431.3%
    2. Neuroscience53.3%
    1. Medicine107.4%
    1. Psychology85.5%
  1. 1Genetics22% of their works
  2. 2Molecular Biology9% of their works
  3. 3Experimental and Cognitive Psychology6% of their works
  4. 4Neurology4% of their works
  5. 5Epidemiology3% of their works
  6. 6Cognitive Neuroscience3% of their works

Research topics

Which keywords describe Matthew H. McIntyre's research?

The keywords of their largest research topics: Genetic Variation, Genome-Wide Association, a-Synuclein, Parkinson's Disease, Congenital Adrenal Hyperplasia, Evolutionary Psychology, Human Behavior and Steroid 21-Hydroxylase Deficiency.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 16 words, with their numbers
  1. Genetic Variation361 topic
  2. Genome-Wide Association361 topic
  3. Parkinson's Disease111 topic
  4. a-Synuclein111 topic
  5. Congenital Adrenal Hyperplasia101 topic
  6. Evolutionary Psychology101 topic
  7. Human Behavior101 topic
  8. Steroid 21-Hydroxylase Deficiency101 topic
  9. Fragile X Syndrome71 topic
  10. Rett Syndrome71 topic
  11. BRCA161 topic
  12. BRCA261 topic
  13. Copy Number Variation61 topic
  14. Genomic Rearrangements61 topic
  15. Sex Determination51 topic
  16. X Chromosome Inactivation51 topic

Where does Matthew H. McIntyre work?

Matthew H. McIntyre's main affiliation in the publication record is 23andMe (United States), United States.

How many publications and citations does Matthew H. McIntyre have?

OpenAlex counts 139 works and 13,995 citations for Matthew H. McIntyre, an h-index of 51, ranking #14,507 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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