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Researcher · University of Exeter · United Kingdom

Michael N. Weedon

Michael N. Weedon publishes mostly in Genetics, Surgery and Molecular Biology, on topics such as Genetic Associations and Epidemiology, Pancreatic function and diabetes and Diabetes and associated disorders.

ORCID 0000-0002-6174-6135 OpenAlex
World rank
#11,067
of 7,710,434 ranked researchers
Rank in United Kingdom
#732
of 367,258
Works
478
Citations
76k
Citations per work
159.8
h-index
106
i10-index 252Top 0.5% in Genetics (Biochemistry, Genetics and Molecular Biology)
Michael N. Weedon

What does Michael N. Weedon research?

Michael N. Weedon's work is mostly in Biochemistry, Genetics and Molecular Biology (45%), Medicine (22%), Psychology (4%).

Life Sciences: 216 works · 45.1% of their workBiochemistry, Genetics and Molecular Biology: 216 works · 45.1% of their workGenetics: 164 works · 34.4% of their workMolecular Biology: 51 works · 10.7% of their workEverything else: 136 works · 28.4% of their workHealth Sciences: 106 works · 22.2% of their workMedicine: 106 works · 22.2% of their workSurgery: 53 works · 11.0% of their workEndocrinology, Diabetes and Metabolism: 42 works · 8.7% of their workPhysiology: 12 works · 2.5% of their workSocial Sciences: 21 works · 4.3% of their workPsychology: 21 works · 4.3% of their workExperimental and Cognitive Psychology: 21 works · 4.3% of their workTHEIR WORKMichael N. Weedon478works
Life Sciences45%Health Sciences22%Social Sciences4%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology21645.1%
  2. Everything else13628.4%
    1. Medicine10622.2%
    1. Psychology214.3%
  1. 1Genetics34% of their works
  2. 2Surgery11% of their works
  3. 3Molecular Biology11% of their works
  4. 4Endocrinology, Diabetes and Metabolism9% of their works
  5. 5Experimental and Cognitive Psychology4% of their works
  6. 6Physiology2% of their works

Research topics

Which keywords describe Michael N. Weedon's research?

The keywords of their largest research topics: Genetic Variation, Genome-Wide Association, Haplotype Mapping, Epidemiology, Pancreatic Islets, ß-Cell Dysfunction, Type 1 Diabetes and Autoimmunity.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Genetic Variation1872 topics
  2. Genome-Wide Association1611 topic
  3. Haplotype Mapping1611 topic
  4. Epidemiology1242 topics
  5. Pancreatic Islets1191 topic
  6. ß-Cell Dysfunction1191 topic
  7. Type 1 Diabetes1182 topics
  8. Autoimmunity821 topic
  9. Genetics821 topic
  10. Glycemic Control582 topics
  11. Insulin Therapy582 topics
  12. Genetic Variants481 topic
  13. Sequence Interpretation481 topic
  14. Insomnia421 topic
  15. Sleep Duration421 topic
  16. Diabetes361 topic
  17. Hypoglycemia361 topic
  18. Nutrigenomics261 topic
  19. Personalized Nutrition261 topic
  20. Cardiovascular Outcomes221 topic
  21. Type 2 Diabetes221 topic

Where does Michael N. Weedon work?

Michael N. Weedon's main affiliation in the publication record is University of Exeter, United Kingdom.

How many publications and citations does Michael N. Weedon have?

OpenAlex counts 478 works and 76,362 citations for Michael N. Weedon, an h-index of 106, ranking #11,067 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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