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Researcher · University of Bonn · Germany

Michael Thomas Heneka

Michael Thomas Heneka publishes mostly in Neurology, Physiology and Molecular Biology, on topics such as Alzheimer's disease research and treatments, Neuroinflammation and Neurodegeneration Mechanisms and Dementia and Cognitive Impairment Research.

ORCID 0000-0003-4996-1630 OpenAlex
World rank
#29,361
of 7,710,434 ranked researchers
Rank in Germany
#1,713
of 364,343
Works
587
Citations
58k
Citations per work
98.1
h-index
115
i10-index 325Top 0.1% in Neurology (Neuroscience)
Michael Thomas Heneka

What does Michael Thomas Heneka research?

Michael Thomas Heneka's work is mostly in Medicine (25%), Neuroscience (21%), Biochemistry, Genetics and Molecular Biology (11%).

Everything else: 223 works · 38.0% of their workLife Sciences: 217 works · 36.9% of their workNeuroscience: 124 works · 21.1% of their workNeurology: 97 works · 16.6% of their workCognitive Neuroscience: 26 works · 4.5% of their workBiochemistry, Genetics and Molecular Biology: 64 works · 10.9% of their workMolecular Biology: 64 works · 10.9% of their workImmunology and Microbiology: 29 works · 4.9% of their workImmunology: 29 works · 4.9% of their workHealth Sciences: 147 works · 25.1% of their workMedicine: 147 works · 25.1% of their workPhysiology: 96 works · 16.4% of their workPsychiatry and Mental health: 51 works · 8.7% of their workTHEIR WORKMichael ThomasHeneka587works
Life Sciences37%Health Sciences25%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else22338.0%
    1. Neuroscience12421.1%
    2. Biochemistry, Genetics and Molecular Biology6410.9%
    3. Immunology and Microbiology294.9%
    1. Medicine14725.1%
  1. 1Neurology17% of their works
  2. 2Physiology16% of their works
  3. 3Molecular Biology11% of their works
  4. 4Psychiatry and Mental health9% of their works
  5. 5Immunology5% of their works
  6. 6Cognitive Neuroscience4% of their works

Research topics

Which keywords describe Michael Thomas Heneka's research?

The keywords of their largest research topics: Neuroinflammation, Neurodegeneration, Oxidative Stress, Cytokines, Microglia, Amyloid Hypothesis, Tau Pathology and Astrocytes.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Neuroinflammation5223 topics
  2. Neurodegeneration4552 topics
  3. Oxidative Stress3283 topics
  4. Cytokines2872 topics
  5. Microglia2872 topics
  6. Amyloid Hypothesis2351 topic
  7. Tau Pathology2351 topic
  8. Astrocytes2201 topic
  9. Synaptic Pruning2201 topic
  10. Alzheimer's Disease1341 topic
  11. Mild Cognitive Impairment1341 topic
  12. Inflammation712 topics
  13. Depression671 topic
  14. Tryptophan Metabolism671 topic
  15. Functional Connectivity471 topic
  16. Resting-State fMRI471 topic
  17. PPARs441 topic
  18. Peroxisome Proliferator-Activated Receptors441 topic
  19. Inflammasome271 topic
  20. NLRP3271 topic
  21. Cerebral Hypoperfusion261 topic
  22. Vascular Cognitive Impairment261 topic

Where does Michael Thomas Heneka work?

Michael Thomas Heneka's main affiliation in the publication record is University of Bonn, Germany.

How many publications and citations does Michael Thomas Heneka have?

OpenAlex counts 587 works and 57,572 citations for Michael Thomas Heneka, an h-index of 115, ranking #29,361 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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