Science Explorer Back to mapMap
Discussion
Researcher · University of Gothenburg · Sweden

Nicholas James Ashton

Nicholas James Ashton publishes mostly in Physiology, Psychiatry and Mental health and Neurology, on topics such as Alzheimer's disease research and treatments, Dementia and Cognitive Impairment Research and Neuroinflammation and Neurodegeneration Mechanisms.

ORCID 0000-0002-3579-8804 OpenAlex
World rank
#12,390
of 7,710,434 ranked researchers
Rank in Sweden
#60
of 71,437
Works
687
Citations
30k
Citations per work
44.3
h-index
91
i10-index 335Top 0.5% in Physiology (Medicine)
Nicholas James Ashton

What does Nicholas James Ashton research?

Nicholas James Ashton's work is mostly in Medicine (54%), Neuroscience (7%), Biochemistry, Genetics and Molecular Biology (7%).

Health Sciences: 373 works · 54.3% of their workMedicine: 373 works · 54.3% of their workPhysiology: 164 works · 23.8% of their workPsychiatry and Mental health: 147 works · 21.4% of their workNeurology: 45 works · 6.5% of their workRadiology, Nuclear Medicine and Imaging: 18 works · 2.6% of their workEverything else: 213 works · 31.0% of their workLife Sciences: 101 works · 14.7% of their workNeuroscience: 51 works · 7.4% of their workNeurology: 51 works · 7.4% of their workBiochemistry, Genetics and Molecular Biology: 50 works · 7.3% of their workMolecular Biology: 50 works · 7.3% of their workTHEIR WORKNicholas JamesAshton687works
Life Sciences15%Health Sciences54%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine37354.3%
  2. Everything else21331.0%
    1. Neuroscience517.4%
    2. Biochemistry, Genetics and Molecular Biology507.3%
  1. 1Physiology24% of their works
  2. 2Psychiatry and Mental health21% of their works
  3. 3Neurology7% of their works
  4. 4Molecular Biology7% of their works
  5. 5Neurology6% of their works
  6. 6Radiology, Nuclear Medicine and Imaging3% of their works

Research topics

Which keywords describe Nicholas James Ashton's research?

The keywords of their largest research topics: Neurodegeneration, Neuroinflammation, Oxidative Stress, Amyloid Hypothesis, Tau Pathology, Alzheimer's Disease, Mild Cognitive Impairment and Innate Immunity.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 20 words, with their numbers
  1. Neurodegeneration5413 topics
  2. Neuroinflammation5333 topics
  3. Oxidative Stress4662 topics
  4. Amyloid Hypothesis4241 topic
  5. Tau Pathology4241 topic
  6. Alzheimer's Disease3751 topic
  7. Mild Cognitive Impairment3751 topic
  8. Innate Immunity1172 topics
  9. Astrocytes761 topic
  10. Microglia761 topic
  11. Cerebral Hypoperfusion421 topic
  12. Vascular Cognitive Impairment421 topic
  13. Inflammation411 topic
  14. S100 Proteins411 topic
  15. Chemotherapy351 topic
  16. Cognitive Function351 topic
  17. Cerebral Amyloid Angiopathy331 topic
  18. Intracerebral Hemorrhage331 topic
  19. Functional Connectivity321 topic
  20. Resting-State fMRI321 topic

Where does Nicholas James Ashton work?

Nicholas James Ashton's main affiliation in the publication record is University of Gothenburg, Sweden.

How many publications and citations does Nicholas James Ashton have?

OpenAlex counts 687 works and 30,436 citations for Nicholas James Ashton, an h-index of 91, ranking #12,390 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Nicholas James Ashton. Posts are plain text and can be reported.