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Researcher · Johnson University · United States

Richard E. Heikkila

Richard E. Heikkila publishes mostly in Neurology, Cellular and Molecular Neuroscience and Molecular Biology, on topics such as Parkinson's Disease Mechanisms and Treatments, Neuroscience and Neuropharmacology Research and Neurotransmitter Receptor Influence on Behavior.

OpenAlex
World rank
#181,788
of 7,710,434 ranked researchers
Rank in United States
#67,833
of 1,627,098
Works
155
Citations
14k
Citations per work
90.1
h-index
54
i10-index 125Top 2.6% in Neurology (Medicine)
Richard E. Heikkila

What does Richard E. Heikkila research?

Richard E. Heikkila's work is mostly in Medicine (34%), Neuroscience (20%), Biochemistry, Genetics and Molecular Biology (10%).

Health Sciences: 52 works · 33.6% of their workMedicine: 52 works · 33.6% of their workNeurology: 37 works · 23.9% of their workPediatrics, Perinatology and Child Health: 8 works · 5.4% of their workPhysiology: 7 works · 4.3% of their workEverything else: 48 works · 31.2% of their workLife Sciences: 47 works · 30.2% of their workNeuroscience: 32 works · 20.5% of their workCellular and Molecular Neuroscience: 32 works · 20.5% of their workBiochemistry, Genetics and Molecular Biology: 15 works · 9.7% of their workMolecular Biology: 15 works · 9.7% of their workPhysical Sciences: 8 works · 5.0% of their workChemistry: 8 works · 5.0% of their workOrganic Chemistry: 8 works · 5.0% of their workTHEIR WORKRichard E.Heikkila155works
Life Sciences30%Physical Sciences5%Health Sciences34%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine5233.6%
  2. Everything else4831.2%
    1. Neuroscience3220.5%
    2. Biochemistry, Genetics and Molecular Biology159.7%
    1. Chemistry85.0%
  1. 1Neurology24% of their works
  2. 2Cellular and Molecular Neuroscience20% of their works
  3. 3Molecular Biology10% of their works
  4. 4Pediatrics, Perinatology and Child Health5% of their works
  5. 5Organic Chemistry5% of their works
  6. 6Physiology4% of their works

Research topics

Which keywords describe Richard E. Heikkila's research?

The keywords of their largest research topics: Parkinson's Disease, Oxidative Stress, Neurological Disorders, a-Synuclein, Neurodegeneration, Neurotransmission, Synaptic Plasticity and Dopamine.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Parkinson's Disease892 topics
  2. Oxidative Stress782 topics
  3. Neurological Disorders773 topics
  4. Neurodegeneration661 topic
  5. a-Synuclein661 topic
  6. Neurotransmission401 topic
  7. Synaptic Plasticity401 topic
  8. Dopamine341 topic
  9. Serotonin341 topic
  10. Deep Brain Stimulation231 topic
  11. Subthalamic Nucleus231 topic
  12. Botulinum Toxin141 topic
  13. Neurotoxins141 topic
  14. Antiepileptic Drugs121 topic
  15. Prenatal Exposure121 topic
  16. Exposure81 topic
  17. Pesticides81 topic
  18. Electrochemical Biosensors71 topic
  19. Graphene71 topic

Where does Richard E. Heikkila work?

Richard E. Heikkila's main affiliation in the publication record is Johnson University, United States.

How many publications and citations does Richard E. Heikkila have?

OpenAlex counts 155 works and 13,958 citations for Richard E. Heikkila, an h-index of 54, ranking #181,788 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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