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Researcher · Hertie Institute for Clinical Brain Research · Germany

Sarah K. Fritschi

Sarah K. Fritschi publishes mostly in Physiology, Molecular Biology and Neurology, on topics such as Alzheimer's disease research and treatments, Prion Diseases and Protein Misfolding and Amyloidosis: Diagnosis, Treatment, Outcomes.

ORCID 0000-0003-4665-7203 OpenAlex
World rank
#1,875,326
of 7,710,434 ranked researchers
Rank in Germany
#92,526
of 364,343
Works
20
Citations
1.7k
Citations per work
82.9
h-index
12
i10-index 12Top 50% in Physiology (Medicine)
Sarah K. Fritschi

What does Sarah K. Fritschi research?

Sarah K. Fritschi's work is mostly in Medicine (44%), Biochemistry, Genetics and Molecular Biology (24%), Neuroscience (11%).

Health Sciences: 9 works · 44.5% of their workMedicine: 9 works · 44.5% of their workPhysiology: 7 works · 35.2% of their workNeurology: 2 works · 9.3% of their workLife Sciences: 7 works · 35.2% of their workBiochemistry, Genetics and Molecular Biology: 5 works · 24.1% of their workMolecular Biology: 5 works · 24.1% of their workNeuroscience: 2 works · 11.1% of their workNeurology: 1 works · 7.4% of their workCognitive Neuroscience: 1 works · 3.7% of their workEverything else: 3 works · 14.7% of their workSocial Sciences: 1 works · 5.6% of their workSocial Sciences: 1 works · 5.6% of their workSociology and Political Science: 1 works · 5.6% of their workTHEIR WORKSarah K. Fritschi20works
Life Sciences35%Health Sciences44%Social Sciences6%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine944.5%
    1. Biochemistry, Genetics and Molecular Biology524.1%
    2. Neuroscience211.1%
  3. Everything else314.7%
    1. Social Sciences15.6%
  1. 1Physiology35% of their works
  2. 2Molecular Biology24% of their works
  3. 3Neurology9% of their works
  4. 4Neurology7% of their works
  5. 5Sociology and Political Science6% of their works
  6. 6Cognitive Neuroscience4% of their works

Research topics

Which keywords describe Sarah K. Fritschi's research?

The keywords of their largest research topics: Neurodegeneration, Neuroinflammation, Oxidative Stress, Amyloid Hypothesis, Tau Pathology, Molecular Basis, Prion Diseases and Mitochondrial Dysfunction.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Neurodegeneration254 topics
  2. Neuroinflammation233 topics
  3. Oxidative Stress212 topics
  4. Amyloid Hypothesis191 topic
  5. Tau Pathology191 topic
  6. Molecular Basis101 topic
  7. Prion Diseases101 topic
  8. Mitochondrial Dysfunction42 topics
  9. Neuroimaging42 topics
  10. Cardiac Amyloidosis31 topic
  11. Transthyretin31 topic
  12. Alzheimer's Disease21 topic
  13. Astrocytes21 topic
  14. Brain Iron Accumulation21 topic
  15. Cerebral Amyloid Angiopathy21 topic
  16. Intracerebral Hemorrhage21 topic
  17. Microglia21 topic
  18. Mild Cognitive Impairment21 topic
  19. Pantothenate Kinase21 topic
  20. Parkinson's Disease21 topic
  21. a-Synuclein21 topic

Where does Sarah K. Fritschi work?

Sarah K. Fritschi's main affiliation in the publication record is Hertie Institute for Clinical Brain Research, Germany.

How many publications and citations does Sarah K. Fritschi have?

OpenAlex counts 20 works and 1,658 citations for Sarah K. Fritschi, an h-index of 12, ranking #1,875,326 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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