Socrates J. Tzartos
Socrates J. Tzartos publishes mostly in Neurology, Molecular Biology and Radiology, Nuclear Medicine and Imaging, on topics such as Myasthenia Gravis and Thymoma, Monoclonal and Polyclonal Antibodies Research and Peripheral Neuropathies and Disorders.
- World rank
- #603,883 of 7,710,434 ranked researchers
- Rank in Greece
- #2,186 of 33,280
- Works
- 314
- Citations
- 12k
- Citations per work
- 37.0
What does Socrates J. Tzartos research?
Socrates J. Tzartos's work is mostly in Medicine (46%), Biochemistry, Genetics and Molecular Biology (24%), Neuroscience (2%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Health Sciences14546.3%
- Medicine14546.3%
- Life Sciences8727.8%
- Everything else8125.9%
- 1Neurology30% of their works
- 2Molecular Biology24% of their works
- 3Radiology, Nuclear Medicine and Imaging12% of their works
- 4Infectious Diseases4% of their works
- 5Cellular and Molecular Neuroscience2% of their works
- 6Immunology2% of their works
Research topics
- Myasthenia Gravis and Thymoma
- Monoclonal and Polyclonal Antibodies Research
- Peripheral Neuropathies and Disorders
- Nicotinic Acetylcholine Receptors Study
- Ion channel regulation and function
- Receptor Mechanisms and Signaling
- Antifungal resistance and susceptibility
- Autoimmune Neurological Disorders and Treatments
Which keywords describe Socrates J. Tzartos's research?
The keywords of their largest research topics: Prognosis, Epidemiology, Autoantibodies, Immunotherapy, Myasthenia Gravis, Thymic Tumors, Crystal Structure and Fc Receptors.
- Anti-NMDA Receptor Encephalitis
- Candidiasis
- G-Protein-Coupled Receptors
- Ion Channels
- Neuronal Signaling
- Chronic Inflammatory Demyelinating Polyradiculoneuropathy
- Neurological Disorders
- Fc Receptors
- Thymic Tumors
- Immunotherapy
- Epidemiology
- Prognosis
- Autoantibodies
- Myasthenia Gravis
- Crystal Structure
- Therapeutic Antibodies
- Cell Signaling
- Guillain-Barré Syndrome
- Nicotinic Acetylcholine Receptors
- Voltage-Gated Channels
- Signal Transduction
- Invasive Fungal Infections
- Paraneoplastic Syndromes
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 23 words, with their numbers
- Prognosis2212 topics
- Epidemiology1742 topics
- Autoantibodies1592 topics
- Immunotherapy1592 topics
- Myasthenia Gravis1401 topic
- Thymic Tumors1401 topic
- Crystal Structure1112 topics
- Fc Receptors1061 topic
- Therapeutic Antibodies1061 topic
- Neurological Disorders1002 topics
- Cell Signaling832 topics
- Chronic Inflammatory Demyelinating Polyradiculoneuropathy811 topic
- Guillain-Barré Syndrome811 topic
- Neuronal Signaling711 topic
- Nicotinic Acetylcholine Receptors711 topic
- Ion Channels431 topic
- Voltage-Gated Channels431 topic
- G-Protein-Coupled Receptors401 topic
- Signal Transduction401 topic
- Candidiasis341 topic
- Invasive Fungal Infections341 topic
- Anti-NMDA Receptor Encephalitis191 topic
- Paraneoplastic Syndromes191 topic
Where does Socrates J. Tzartos work?
Socrates J. Tzartos's main affiliation in the publication record is Pasteur Hellenic Institute, Greece.
How many publications and citations does Socrates J. Tzartos have?
OpenAlex counts 314 works and 11,616 citations for Socrates J. Tzartos, an h-index of 54, ranking #603,883 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
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