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Researcher · deCODE Genetics (Iceland) · Iceland

Thorgeir Elís Thorgeirsson

Thorgeir Elís Thorgeirsson publishes mostly in Genetics, Molecular Biology and Cellular and Molecular Neuroscience, on topics such as Genetic Associations and Epidemiology, Genetics and Neurodevelopmental Disorders and Epigenetics and DNA Methylation.

ORCID 0000-0002-5149-7040 OpenAlex
World rank
#22,049
of 7,710,434 ranked researchers
Rank in Iceland
#24
of 2,050
Works
180
Citations
38k
Citations per work
210.9
h-index
70
i10-index 134Top 1.9% in Genetics (Biochemistry, Genetics and Molecular Biology)
Thorgeir Elís Thorgeirsson

What does Thorgeir Elís Thorgeirsson research?

Thorgeir Elís Thorgeirsson's work is mostly in Biochemistry, Genetics and Molecular Biology (42%), Medicine (9%), Neuroscience (6%).

Life Sciences: 86 works · 47.7% of their workBiochemistry, Genetics and Molecular Biology: 75 works · 41.9% of their workGenetics: 51 works · 28.1% of their workMolecular Biology: 25 works · 13.8% of their workNeuroscience: 10 works · 5.8% of their workCellular and Molecular Neuroscience: 10 works · 5.8% of their workEverything else: 73 works · 40.7% of their workHealth Sciences: 16 works · 8.9% of their workMedicine: 16 works · 8.9% of their workPsychiatry and Mental health: 10 works · 5.6% of their workEpidemiology: 6 works · 3.3% of their workSocial Sciences: 5 works · 2.7% of their workPsychology: 5 works · 2.7% of their workClinical Psychology: 5 works · 2.7% of their workTHEIR WORKThorgeir ElísThorgeirsson180works
Life Sciences48%Health Sciences9%Social Sciences3%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology7541.9%
    2. Neuroscience105.8%
  2. Everything else7340.7%
    1. Medicine168.9%
    1. Psychology52.7%
  1. 1Genetics28% of their works
  2. 2Molecular Biology14% of their works
  3. 3Cellular and Molecular Neuroscience6% of their works
  4. 4Psychiatry and Mental health6% of their works
  5. 5Epidemiology3% of their works
  6. 6Clinical Psychology3% of their works

Research topics

Which keywords describe Thorgeir Elís Thorgeirsson's research?

The keywords of their largest research topics: Genetic Variation, Genome-Wide Association, DNA Methylation, Fragile X Syndrome, Histone Modification, Rett Syndrome, Channelrhodopsin and Optogenetics.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 16 words, with their numbers
  1. Genetic Variation681 topic
  2. Genome-Wide Association681 topic
  3. DNA Methylation181 topic
  4. Fragile X Syndrome181 topic
  5. Histone Modification181 topic
  6. Rett Syndrome181 topic
  7. Channelrhodopsin111 topic
  8. Optogenetics111 topic
  9. Bipolar Spectrum Disorder101 topic
  10. Prevalence101 topic
  11. Copy Number Variation91 topic
  12. Genetic Variants91 topic
  13. Genomic Rearrangements91 topic
  14. Headache Disorders91 topic
  15. Migraine91 topic
  16. Sequence Interpretation91 topic

Where does Thorgeir Elís Thorgeirsson work?

Thorgeir Elís Thorgeirsson's main affiliation in the publication record is deCODE Genetics (Iceland), Iceland.

How many publications and citations does Thorgeir Elís Thorgeirsson have?

OpenAlex counts 180 works and 37,956 citations for Thorgeir Elís Thorgeirsson, an h-index of 70, ranking #22,049 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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