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Researcher · Saarland University · Germany

Veit Flockerzi

Veit Flockerzi publishes mostly in Molecular Biology, Sensory Systems and Cellular and Molecular Neuroscience, on topics such as Ion channel regulation and function, Ion Channels and Receptors and Cardiac electrophysiology and arrhythmias.

ORCID 0000-0001-6002-6294 OpenAlex
World rank
#103,459
of 7,710,434 ranked researchers
Rank in Germany
#5,513
of 364,343
Works
329
Citations
27k
Citations per work
83.4
h-index
86
i10-index 226Top 0.5% in Molecular Biology
Veit Flockerzi

What does Veit Flockerzi research?

Veit Flockerzi's work is mostly in Biochemistry, Genetics and Molecular Biology (30%), Neuroscience (29%), Medicine (9%).

Life Sciences: 201 works · 61.2% of their workBiochemistry, Genetics and Molecular Biology: 99 works · 30.0% of their workMolecular Biology: 99 works · 30.0% of their workNeuroscience: 95 works · 28.8% of their workSensory Systems: 55 works · 16.8% of their workCellular and Molecular Neuroscience: 39 works · 12.0% of their workImmunology and Microbiology: 8 works · 2.4% of their workImmunology: 8 works · 2.4% of their workEverything else: 83 works · 25.3% of their workHealth Sciences: 44 works · 13.5% of their workMedicine: 29 works · 8.9% of their workCardiology and Cardiovascular Medicine: 29 works · 8.9% of their workNursing: 15 works · 4.6% of their workNutrition and Dietetics: 15 works · 4.6% of their workTHEIR WORKVeit Flockerzi329works
Life Sciences61%Health Sciences14%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology9930.0%
    2. Neuroscience9528.8%
    3. Immunology and Microbiology82.4%
  2. Everything else8325.3%
    1. Medicine298.9%
    2. Nursing154.6%
  1. 1Molecular Biology30% of their works
  2. 2Sensory Systems17% of their works
  3. 3Cellular and Molecular Neuroscience12% of their works
  4. 4Cardiology and Cardiovascular Medicine9% of their works
  5. 5Nutrition and Dietetics5% of their works
  6. 6Immunology2% of their works

Research topics

Which keywords describe Veit Flockerzi's research?

The keywords of their largest research topics: Ion Channels, Calcium Signaling, Calcium Channels, Voltage-Gated Channels, Sensory Neurons, TRP Channels, Neuronal Circuits and Arrhythmias.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Ion Channels3573 topics
  2. Calcium Signaling2032 topics
  3. Calcium Channels1541 topic
  4. Voltage-Gated Channels1541 topic
  5. Sensory Neurons1361 topic
  6. TRP Channels1361 topic
  7. Neuronal Circuits912 topics
  8. Arrhythmias671 topic
  9. Long-QT Syndrome671 topic
  10. Neurotransmission491 topic
  11. Synaptic Plasticity491 topic
  12. Drosophila421 topic
  13. Neuroscience421 topic
  14. Crystal Structure261 topic
  15. Nicotinic Acetylcholine Receptors261 topic
  16. Sweet Taste231 topic
  17. Taste Receptors231 topic
  18. Lysosomal Calcium Signaling171 topic
  19. Two-pore Channels171 topic

Where does Veit Flockerzi work?

Veit Flockerzi's main affiliation in the publication record is Saarland University, Germany.

How many publications and citations does Veit Flockerzi have?

OpenAlex counts 329 works and 27,438 citations for Veit Flockerzi, an h-index of 86, ranking #103,459 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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