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Researcher · 23andMe (United States) · United States

Adam Auton

Adam Auton publishes mostly in Genetics, Molecular Biology and Epidemiology, on topics such as Genetic Associations and Epidemiology, Genetic Mapping and Diversity in Plants and Animals and Genetic and phenotypic traits in livestock.

ORCID 0000-0002-1630-1225 OpenAlex
World rank
#1,470
of 7,710,434 ranked researchers
Rank in United States
#589
of 1,627,098
Works
181
Citations
105k
Citations per work
580.2
h-index
81
i10-index 130Top 1.2% in Genetics (Biochemistry, Genetics and Molecular Biology)
Adam Auton

What does Adam Auton research?

Adam Auton's work is mostly in Biochemistry, Genetics and Molecular Biology (60%), Medicine (5%), Psychology (3%).

Life Sciences: 108 works · 59.8% of their workBiochemistry, Genetics and Molecular Biology: 108 works · 59.8% of their workGenetics: 81 works · 44.9% of their workMolecular Biology: 24 works · 13.1% of their workCancer Research: 3 works · 1.8% of their workEverything else: 58 works · 32.3% of their workHealth Sciences: 10 works · 5.4% of their workMedicine: 10 works · 5.4% of their workEpidemiology: 5 works · 2.9% of their workSurgery: 5 works · 2.5% of their workSocial Sciences: 5 works · 2.5% of their workPsychology: 5 works · 2.5% of their workExperimental and Cognitive Psychology: 5 works · 2.5% of their workTHEIR WORKAdam Auton181works
Life Sciences60%Health Sciences5%Social Sciences3%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology10859.8%
  2. Everything else5832.3%
    1. Medicine105.4%
    1. Psychology52.5%
  1. 1Genetics45% of their works
  2. 2Molecular Biology13% of their works
  3. 3Epidemiology3% of their works
  4. 4Surgery2% of their works
  5. 5Experimental and Cognitive Psychology2% of their works
  6. 6Cancer Research2% of their works

Research topics

Which keywords describe Adam Auton's research?

The keywords of their largest research topics: Population Genetics, Genetic Variation, Genome-Wide Association, Haplotype Mapping, Mendelian Randomization, Marker-Assisted Selection, Genome-wide Association Studies and Quantitative Trait Loci.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 20 words, with their numbers
  1. Population Genetics1003 topics
  2. Genetic Variation992 topics
  3. Genome-Wide Association902 topics
  4. Haplotype Mapping741 topic
  5. Mendelian Randomization741 topic
  6. Marker-Assisted Selection412 topics
  7. Genome-wide Association Studies251 topic
  8. Quantitative Trait Loci251 topic
  9. Genomic Selection161 topic
  10. Plant Breeding161 topic
  11. Genetic Variants151 topic
  12. Sequence Interpretation151 topic
  13. Copy Number Variation141 topic
  14. Genomic Rearrangements141 topic
  15. RNA-Seq121 topic
  16. sequence alignment121 topic
  17. Fragile X Syndrome111 topic
  18. Rett Syndrome111 topic
  19. Adaptation101 topic
  20. Evolution101 topic

Where does Adam Auton work?

Adam Auton's main affiliation in the publication record is 23andMe (United States), United States.

How many publications and citations does Adam Auton have?

OpenAlex counts 181 works and 105,017 citations for Adam Auton, an h-index of 81, ranking #1,470 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Other researchers named Adam Auton

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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