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Researcher · Universidad de Salamanca · Spain

Alicia Rodríguez‐Barbero

Alicia Rodríguez‐Barbero publishes mostly in Molecular Biology, Cardiology and Cardiovascular Medicine and Pharmacology, on topics such as Nitric Oxide and Endothelin Effects, TGF-β signaling in diseases and Angiogenesis and VEGF in Cancer.

ORCID 0000-0002-6774-7147 OpenAlex
World rank
#3,596,039
of 7,710,434 ranked researchers
Rank in Spain
#81,409
of 204,306
Works
98
Citations
2.4k
Citations per work
24.7
h-index
28
i10-index 51Top 17% in Molecular Biology
Alicia Rodríguez‐Barbero

What does Alicia Rodríguez‐Barbero research?

Alicia Rodríguez‐Barbero's work is mostly in Medicine (22%), Biochemistry, Genetics and Molecular Biology (18%), Neuroscience (4%).

Everything else: 54 works · 55.5% of their workHealth Sciences: 22 works · 22.4% of their workMedicine: 22 works · 22.4% of their workCardiology and Cardiovascular Medicine: 6 works · 6.6% of their workPharmacology: 5 works · 5.4% of their workPhysiology: 5 works · 5.4% of their workOncology: 5 works · 5.0% of their workLife Sciences: 22 works · 22.1% of their workBiochemistry, Genetics and Molecular Biology: 18 works · 18.2% of their workMolecular Biology: 18 works · 18.2% of their workNeuroscience: 4 works · 3.9% of their workCellular and Molecular Neuroscience: 4 works · 3.9% of their workTHEIR WORKAliciaRodríguez‐Barbero98works
Life Sciences22%Health Sciences22%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else5455.5%
    1. Medicine2222.4%
    1. Biochemistry, Genetics and Molecular Biology1818.2%
    2. Neuroscience43.9%
  1. 1Molecular Biology18% of their works
  2. 2Cardiology and Cardiovascular Medicine7% of their works
  3. 3Pharmacology5% of their works
  4. 4Physiology5% of their works
  5. 5Oncology5% of their works
  6. 6Cellular and Molecular Neuroscience4% of their works

Research topics

Which keywords describe Alicia Rodríguez‐Barbero's research?

The keywords of their largest research topics: Angiogenesis, Cancer Progression, Inflammation, Endothelial Dysfunction, Nitric Oxide, Smad-dependent pathways, Tumor Suppression and endothelial progenitor cells.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Angiogenesis182 topics
  2. Cancer Progression182 topics
  3. Inflammation182 topics
  4. Endothelial Dysfunction121 topic
  5. Nitric Oxide121 topic
  6. Smad-dependent pathways111 topic
  7. Tumor Suppression111 topic
  8. Ivermectin81 topic
  9. Lymphatic Filariasis81 topic
  10. VEGF81 topic
  11. endothelial progenitor cells81 topic
  12. Antibiotic Pharmacokinetics71 topic
  13. CCN Family71 topic
  14. Connective Tissue Growth Factor71 topic
  15. Critically Ill Patients71 topic
  16. Cancer61 topic
  17. Cyclooxygenase-2 Inhibitors61 topic
  18. Diagnosis61 topic
  19. Hereditary Hemorrhagic Telangiectasia61 topic

Where does Alicia Rodríguez‐Barbero work?

Alicia Rodríguez‐Barbero's main affiliation in the publication record is Universidad de Salamanca, Spain.

How many publications and citations does Alicia Rodríguez‐Barbero have?

OpenAlex counts 98 works and 2,422 citations for Alicia Rodríguez‐Barbero, an h-index of 28, ranking #3,596,039 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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