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Researcher · Paracelsus Elena Klinik Kassel · Germany

Brit Mollenhauer

Brit Mollenhauer publishes mostly in Neurology, Physiology and Molecular Biology, on topics such as Parkinson's Disease Mechanisms and Treatments, Neurological disorders and treatments and Alzheimer's disease research and treatments.

ORCID 0000-0001-8437-3645 OpenAlex
World rank
#18,300
of 7,710,434 ranked researchers
Rank in Germany
#1,062
of 364,343
Works
478
Citations
42k
Citations per work
87.7
h-index
93
i10-index 283Top 0.4% in Neurology (Medicine)
Brit Mollenhauer

What does Brit Mollenhauer research?

Brit Mollenhauer's work is mostly in Medicine (57%), Neuroscience (9%), Biochemistry, Genetics and Molecular Biology (9%).

Health Sciences: 272 works · 57.0% of their workMedicine: 272 works · 57.0% of their workNeurology: 202 works · 42.3% of their workPhysiology: 47 works · 9.8% of their workPsychiatry and Mental health: 23 works · 4.9% of their workEverything else: 120 works · 25.1% of their workLife Sciences: 86 works · 17.9% of their workNeuroscience: 43 works · 9.0% of their workCellular and Molecular Neuroscience: 23 works · 4.9% of their workNeurology: 20 works · 4.1% of their workBiochemistry, Genetics and Molecular Biology: 43 works · 8.9% of their workMolecular Biology: 43 works · 8.9% of their workTHEIR WORKBrit Mollenhauer478works
Life Sciences18%Health Sciences57%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine27257.0%
  2. Everything else12025.1%
    1. Neuroscience439.0%
    2. Biochemistry, Genetics and Molecular Biology438.9%
  1. 1Neurology42% of their works
  2. 2Physiology10% of their works
  3. 3Molecular Biology9% of their works
  4. 4Cellular and Molecular Neuroscience5% of their works
  5. 5Psychiatry and Mental health5% of their works
  6. 6Neurology4% of their works

Research topics

Which keywords describe Brit Mollenhauer's research?

The keywords of their largest research topics: Parkinson's Disease, Neurodegeneration, Oxidative Stress, Dopaminergic Neurons, Mitochondrial Dysfunction, Diagnostic Criteria, a-Synuclein and Genetic Risk Factors.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 24 words, with their numbers
  1. Parkinson's Disease5073 topics
  2. Neurodegeneration4875 topics
  3. Oxidative Stress3912 topics
  4. Dopaminergic Neurons3552 topics
  5. Mitochondrial Dysfunction3552 topics
  6. Diagnostic Criteria3512 topics
  7. Genetic Risk Factors3201 topic
  8. a-Synuclein3201 topic
  9. Neurological Disorders1832 topics
  10. Deep Brain Stimulation1521 topic
  11. Subthalamic Nucleus1521 topic
  12. Neuroimaging882 topics
  13. Amyloid Hypothesis711 topic
  14. Tau Pathology711 topic
  15. Alzheimer's Disease531 topic
  16. Mild Cognitive Impairment531 topic
  17. Brain Iron Accumulation351 topic
  18. NR4A2351 topic
  19. Nurr1351 topic
  20. Pantothenate Kinase351 topic
  21. Epidemiology311 topic
  22. Restless Legs Syndrome311 topic
  23. FUS/TLS261 topic
  24. TDP-43261 topic

Where does Brit Mollenhauer work?

Brit Mollenhauer's main affiliation in the publication record is Paracelsus Elena Klinik Kassel, Germany.

How many publications and citations does Brit Mollenhauer have?

OpenAlex counts 478 works and 41,910 citations for Brit Mollenhauer, an h-index of 93, ranking #18,300 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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