Cid Czarina E. Diesta
Cid Czarina E. Diesta publishes mostly in Neurology, Cellular and Molecular Neuroscience and Molecular Biology, on topics such as Neurological disorders and treatments, Genetic Neurodegenerative Diseases and Parkinson's Disease Mechanisms and Treatments.
- World rank
- #3,800,718 of 7,710,434 ranked researchers
- Rank in Philippines
- #1,948 of 6,138
- Works
- 67
- Citations
- 873
- Citations per work
- 13.0
What does Cid Czarina E. Diesta research?
Cid Czarina E. Diesta's work is mostly in Medicine (51%), Neuroscience (23%), Biochemistry, Genetics and Molecular Biology (11%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Health Sciences3451.3%
- Medicine3451.3%
- Life Sciences2334.7%
- Everything else811.9%
- Social Sciences12.1%
- Psychology12.1%
- 1Neurology51% of their works
- 2Cellular and Molecular Neuroscience18% of their works
- 3Molecular Biology9% of their works
- 4Neurology6% of their works
- 5Genetics3% of their works
- 6Clinical Psychology2% of their works
Research topics
- Neurological disorders and treatments
- Genetic Neurodegenerative Diseases
- Parkinson's Disease Mechanisms and Treatments
- Botulinum Toxin and Related Neurological Disorders
- Neurological diseases and metabolism
- RNA regulation and disease
- Genetics and Neurodevelopmental Disorders
- Hereditary Neurological Disorders
Which keywords describe Cid Czarina E. Diesta's research?
The keywords of their largest research topics: Parkinson's Disease, Mitochondrial Dysfunction, Neurological Disorders, Deep Brain Stimulation, Subthalamic Nucleus, Neurodegeneration, Huntington's Disease and Protein Aggregation.
- Charcot-Marie-Tooth Disease
- Fragile X Syndrome
- ADAR Deaminases
- Brain Iron Accumulation
- Genetic Mutations
- a-Synuclein
- Protein Aggregation
- Neurodegeneration
- Deep Brain Stimulation
- Mitochondrial Dysfunction
- Parkinson's Disease
- Neurological Disorders
- Subthalamic Nucleus
- Huntington's Disease
- Dopaminergic Neurons
- Botulinum Toxin
- Neurotoxins
- Pantothenate Kinase
- RNA Editing
- Rett Syndrome
- Hereditary Spastic Paraplegia
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 21 words, with their numbers
- Parkinson's Disease732 topics
- Mitochondrial Dysfunction704 topics
- Neurological Disorders653 topics
- Deep Brain Stimulation451 topic
- Subthalamic Nucleus451 topic
- Neurodegeneration362 topics
- Huntington's Disease311 topic
- Protein Aggregation311 topic
- Dopaminergic Neurons281 topic
- a-Synuclein281 topic
- Botulinum Toxin151 topic
- Genetic Mutations153 topics
- Neurotoxins151 topic
- Brain Iron Accumulation81 topic
- Pantothenate Kinase81 topic
- ADAR Deaminases51 topic
- RNA Editing51 topic
- Fragile X Syndrome41 topic
- Rett Syndrome41 topic
- Charcot-Marie-Tooth Disease31 topic
- Hereditary Spastic Paraplegia31 topic
Where does Cid Czarina E. Diesta work?
Cid Czarina E. Diesta's main affiliation in the publication record is Makati Medical Center, Philippines.
How many publications and citations does Cid Czarina E. Diesta have?
OpenAlex counts 67 works and 873 citations for Cid Czarina E. Diesta, an h-index of 18, ranking #3,800,718 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
Discussion
Ask a question or share a note about Cid Czarina E. Diesta. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.