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Researcher · King Abdulaziz University · Saudi Arabia

Ghulam Md Ashraf

Ghulam Md Ashraf publishes mostly in Molecular Biology, Physiology and Pharmacology, on topics such as Alzheimer's disease research and treatments, Computational Drug Discovery Methods and Cholinesterase and Neurodegenerative Diseases.

ORCID 0000-0002-9820-2078 OpenAlex
World rank
#169,204
of 7,710,434 ranked researchers
Rank in Saudi Arabia
#639
of 25,688
Works
430
Citations
16k
Citations per work
36.4
h-index
67
i10-index 277Top 1.4% in Molecular Biology
Ghulam Md Ashraf

What does Ghulam Md Ashraf research?

Ghulam Md Ashraf's work is mostly in Biochemistry, Genetics and Molecular Biology (18%), Medicine (14%), Neuroscience (4%).

Everything else: 244 works · 56.7% of their workLife Sciences: 112 works · 26.1% of their workBiochemistry, Genetics and Molecular Biology: 79 works · 18.3% of their workMolecular Biology: 79 works · 18.3% of their workNeuroscience: 17 works · 4.0% of their workNeurology: 17 works · 4.0% of their workImmunology and Microbiology: 16 works · 3.8% of their workImmunology: 16 works · 3.8% of their workHealth Sciences: 58 works · 13.6% of their workMedicine: 58 works · 13.6% of their workPhysiology: 38 works · 8.9% of their workPharmacology: 20 works · 4.7% of their workPhysical Sciences: 15 works · 3.6% of their workComputer Science: 15 works · 3.6% of their workComputational Theory and Mathematics: 15 works · 3.6% of their workTHEIR WORKGhulam Md Ashraf430works
Life Sciences26%Physical Sciences4%Health Sciences14%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else24456.7%
    1. Biochemistry, Genetics and Molecular Biology7918.3%
    2. Neuroscience174.0%
    3. Immunology and Microbiology163.8%
    1. Medicine5813.6%
    1. Computer Science153.6%
  1. 1Molecular Biology18% of their works
  2. 2Physiology9% of their works
  3. 3Pharmacology5% of their works
  4. 4Neurology4% of their works
  5. 5Immunology4% of their works
  6. 6Computational Theory and Mathematics4% of their works

Research topics

Which keywords describe Ghulam Md Ashraf's research?

The keywords of their largest research topics: Oxidative Stress, Neuroinflammation, Neurodegeneration, Amyloid Hypothesis, Tau Pathology, Neurodegenerative Diseases, Molecular Docking and Virtual Screening.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 22 words, with their numbers
  1. Oxidative Stress1113 topics
  2. Neuroinflammation1083 topics
  3. Neurodegeneration932 topics
  4. Amyloid Hypothesis741 topic
  5. Tau Pathology741 topic
  6. Neurodegenerative Diseases612 topics
  7. Molecular Docking421 topic
  8. Virtual Screening421 topic
  9. Acetylcholinesterase391 topic
  10. Cholinesterase Inhibitors391 topic
  11. Cytokines342 topics
  12. Microglia342 topics
  13. Mitochondria221 topic
  14. Reactive Oxygen Species221 topic
  15. Astrocytes191 topic
  16. Synaptic Pruning191 topic
  17. Depression151 topic
  18. Galectins151 topic
  19. Immune Regulation151 topic
  20. Tryptophan Metabolism151 topic
  21. Drug Delivery141 topic
  22. Nanocarriers141 topic

Where does Ghulam Md Ashraf work?

Ghulam Md Ashraf's main affiliation in the publication record is King Abdulaziz University, Saudi Arabia.

How many publications and citations does Ghulam Md Ashraf have?

OpenAlex counts 430 works and 15,661 citations for Ghulam Md Ashraf, an h-index of 67, ranking #169,204 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Other researchers named Ghulam Md Ashraf

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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