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Researcher · Stavanger University Hospital · Norway

Guido Werner Alves

Guido Werner Alves publishes mostly in Neurology, Molecular Biology and Cellular and Molecular Neuroscience, on topics such as Parkinson's Disease Mechanisms and Treatments, Neurological disorders and treatments and Nuclear Receptors and Signaling.

ORCID 0000-0003-0630-2870 OpenAlex
World rank
#108,633
of 7,710,434 ranked researchers
Rank in Norway
#554
of 33,005
Works
195
Citations
11k
Citations per work
56.0
h-index
55
i10-index 110Top 2.4% in Neurology (Medicine)
Guido Werner Alves

What does Guido Werner Alves research?

Guido Werner Alves's work is mostly in Medicine (62%), Biochemistry, Genetics and Molecular Biology (8%), Neuroscience (8%).

Health Sciences: 120 works · 61.6% of their workMedicine: 120 works · 61.6% of their workNeurology: 89 works · 45.4% of their workPhysiology: 15 works · 7.5% of their workPsychiatry and Mental health: 11 works · 5.7% of their workEpidemiology: 6 works · 3.0% of their workEverything else: 45 works · 23.0% of their workLife Sciences: 30 works · 15.4% of their workBiochemistry, Genetics and Molecular Biology: 15 works · 7.7% of their workMolecular Biology: 15 works · 7.7% of their workNeuroscience: 15 works · 7.7% of their workCellular and Molecular Neuroscience: 15 works · 7.7% of their workTHEIR WORKGuido WernerAlves195works
Life Sciences15%Health Sciences62%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine12061.6%
  2. Everything else4523.0%
    1. Biochemistry, Genetics and Molecular Biology157.7%
    2. Neuroscience157.7%
  1. 1Neurology45% of their works
  2. 2Molecular Biology8% of their works
  3. 3Cellular and Molecular Neuroscience8% of their works
  4. 4Physiology8% of their works
  5. 5Psychiatry and Mental health6% of their works
  6. 6Epidemiology3% of their works

Research topics

Which keywords describe Guido Werner Alves's research?

The keywords of their largest research topics: Parkinson's Disease, Neurodegeneration, Dopaminergic Neurons, Oxidative Stress, a-Synuclein, Genetic Risk Factors, Deep Brain Stimulation and Neurological Disorders.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Parkinson's Disease2536 topics
  2. Neurodegeneration1833 topics
  3. Dopaminergic Neurons1682 topics
  4. Oxidative Stress1612 topics
  5. Genetic Risk Factors1461 topic
  6. a-Synuclein1461 topic
  7. Deep Brain Stimulation622 topics
  8. Neurological Disorders622 topics
  9. Basal Ganglia511 topic
  10. Subthalamic Nucleus511 topic
  11. NR4A2221 topic
  12. Nurr1221 topic
  13. Alzheimer's Disease171 topic
  14. Mild Cognitive Impairment171 topic
  15. Amyloid Hypothesis151 topic
  16. Tau Pathology151 topic
  17. Fall Risk121 topic
  18. Gait Speed121 topic
  19. Camptocormia111 topic
  20. Dropped Head Syndrome111 topic
  21. Enzyme Replacement Therapy111 topic
  22. Lysosomal Storage Disorders111 topic

Where does Guido Werner Alves work?

Guido Werner Alves's main affiliation in the publication record is Stavanger University Hospital, Norway.

How many publications and citations does Guido Werner Alves have?

OpenAlex counts 195 works and 10,921 citations for Guido Werner Alves, an h-index of 55, ranking #108,633 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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