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Researcher · Instituto de Neurología de Buenos Aires · Argentina

Gustavo Da Prat

Gustavo Da Prat publishes mostly in Neurology, Endocrinology, Diabetes and Metabolism and Physiology, on topics such as Neurological disorders and treatments, Parkinson's Disease Mechanisms and Treatments and Neurological and metabolic disorders.

ORCID 0000-0001-7567-9335 OpenAlex
World rank
#6,207,788
of 7,710,434 ranked researchers
Rank in Argentina
#24,510
of 38,473
Works
29
Citations
246
Citations per work
8.5
h-index
7
i10-index 6Top 79% in Neurology (Medicine)
Gustavo Da Prat

What does Gustavo Da Prat research?

Gustavo Da Prat's work is mostly in Medicine (45%), Neuroscience (11%), Biochemistry, Genetics and Molecular Biology (5%).

Health Sciences: 13 works · 45.1% of their workMedicine: 13 works · 45.1% of their workNeurology: 10 works · 32.9% of their workEndocrinology, Diabetes and Metabolism: 2 works · 6.1% of their workPhysiology: 2 works · 6.1% of their workEverything else: 11 works · 39.0% of their workLife Sciences: 5 works · 15.9% of their workNeuroscience: 3 works · 11.0% of their workCellular and Molecular Neuroscience: 2 works · 6.1% of their workNeurology: 1 works · 4.9% of their workBiochemistry, Genetics and Molecular Biology: 1 works · 4.9% of their workGenetics: 1 works · 4.9% of their workTHEIR WORKGustavo Da Prat29works
Life Sciences16%Health Sciences45%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine1345.1%
  2. Everything else1139.0%
    1. Neuroscience311.0%
    2. Biochemistry, Genetics and Molecular Biology14.9%
  1. 1Neurology33% of their works
  2. 2Endocrinology, Diabetes and Metabolism6% of their works
  3. 3Physiology6% of their works
  4. 4Cellular and Molecular Neuroscience6% of their works
  5. 5Genetics5% of their works
  6. 6Neurology5% of their works

Research topics

Which keywords describe Gustavo Da Prat's research?

The keywords of their largest research topics: Parkinson's Disease, Mitochondrial Dysfunction, Neurodegeneration, Basal Ganglia, Deep Brain Stimulation, Neurological Disorders, Epilepsy and Subthalamic Nucleus.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 25 words, with their numbers
  1. Parkinson's Disease233 topics
  2. Mitochondrial Dysfunction173 topics
  3. Neurodegeneration163 topics
  4. Basal Ganglia152 topics
  5. Deep Brain Stimulation142 topics
  6. Neurological Disorders142 topics
  7. Epilepsy132 topics
  8. Subthalamic Nucleus101 topic
  9. Treatment-Resistant Depression101 topic
  10. Dopaminergic Neurons91 topic
  11. Neuroimaging92 topics
  12. a-Synuclein91 topic
  13. Genetic Mutations72 topics
  14. Chorea51 topic
  15. Hyperglycemia51 topic
  16. Brain Iron Accumulation41 topic
  17. Camptocormia41 topic
  18. Dropped Head Syndrome41 topic
  19. Huntington's Disease41 topic
  20. Pantothenate Kinase41 topic
  21. Protein Aggregation41 topic
  22. Glycogen31 topic
  23. Metabolism31 topic
  24. Biochemical Genetics21 topic
  25. Metabolic Disorders21 topic

Where does Gustavo Da Prat work?

Gustavo Da Prat's main affiliation in the publication record is Instituto de Neurología de Buenos Aires, Argentina.

How many publications and citations does Gustavo Da Prat have?

OpenAlex counts 29 works and 246 citations for Gustavo Da Prat, an h-index of 7, ranking #6,207,788 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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