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Researcher · Icahn School of Medicine at Mount Sinai · United States

Helen Vlassara

Helen Vlassara publishes mostly in Clinical Biochemistry, Endocrinology, Diabetes and Metabolism and Molecular Biology, on topics such as Advanced Glycation End Products research, Natural Antidiabetic Agents Studies and Glycosylation and Glycoproteins Research.

OpenAlex
World rank
#5,456
of 7,710,434 ranked researchers
Rank in United States
#1,880
of 1,627,098
Works
261
Citations
41k
Citations per work
157.2
h-index
111
i10-index 198Top 0.2% in Clinical Biochemistry
Helen Vlassara

What does Helen Vlassara research?

Helen Vlassara's work is mostly in Biochemistry, Genetics and Molecular Biology (33%), Medicine (26%), Immunology and Microbiology (4%).

Life Sciences: 98 works · 37.4% of their workBiochemistry, Genetics and Molecular Biology: 86 works · 33.1% of their workClinical Biochemistry: 65 works · 25.0% of their workMolecular Biology: 21 works · 8.1% of their workImmunology and Microbiology: 11 works · 4.3% of their workImmunology: 11 works · 4.3% of their workEverything else: 97 works · 37.0% of their workHealth Sciences: 67 works · 25.6% of their workMedicine: 67 works · 25.6% of their workEndocrinology, Diabetes and Metabolism: 36 works · 13.8% of their workNephrology: 15 works · 5.9% of their workPhysiology: 15 works · 5.9% of their workTHEIR WORKHelen Vlassara261works
Life Sciences37%Health Sciences26%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology8633.1%
    2. Immunology and Microbiology114.3%
  2. Everything else9737.0%
    1. Medicine6725.6%
  1. 1Clinical Biochemistry25% of their works
  2. 2Endocrinology, Diabetes and Metabolism14% of their works
  3. 3Molecular Biology8% of their works
  4. 4Physiology6% of their works
  5. 5Nephrology6% of their works
  6. 6Immunology4% of their works

Research topics

Which keywords describe Helen Vlassara's research?

The keywords of their largest research topics: Oxidative Stress, Diabetes, Inflammation, Advanced Glycation End Products, HMGB1, Insulin Resistance, Antioxidants and Type 2 Diabetes.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Oxidative Stress2724 topics
  2. Diabetes2633 topics
  3. Inflammation2042 topics
  4. Advanced Glycation End Products1861 topic
  5. HMGB11861 topic
  6. Insulin Resistance772 topics
  7. Antioxidants521 topic
  8. Type 2 Diabetes521 topic
  9. Cardiovascular Disease492 topics
  10. Diabetic Nephropathy402 topics
  11. Glycans361 topic
  12. Glycosylation361 topic
  13. Metabolic Syndrome251 topic
  14. Obesity251 topic
  15. Chronic Kidney Disease241 topic
  16. Glomerular Filtration Rate241 topic
  17. Matrix Metalloproteinases191 topic
  18. Tissue Inhibitors of Metalloproteinases191 topic
  19. Alcohol181 topic
  20. Liver Disease181 topic
  21. Carnosine161 topic
  22. ß-Alanine Supplementation161 topic

Where does Helen Vlassara work?

Helen Vlassara's main affiliation in the publication record is Icahn School of Medicine at Mount Sinai, United States.

How many publications and citations does Helen Vlassara have?

OpenAlex counts 261 works and 41,033 citations for Helen Vlassara, an h-index of 111, ranking #5,456 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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