Science Explorer Back to mapMap
Discussion
Researcher · Aichi Human Service Center · Japan

Koh‐ichi Nagata

Koh‐ichi Nagata publishes mostly in Molecular Biology, Cell Biology and Genetics, on topics such as Genetics and Neurodevelopmental Disorders, Protein Kinase Regulation and GTPase Signaling and Cellular transport and secretion.

ORCID 0000-0002-6827-8434 OpenAlex
World rank
#5,280,356
of 7,710,434 ranked researchers
Rank in Japan
#173,476
of 383,136
Works
226
Citations
4.3k
Citations per work
18.8
h-index
35
i10-index 100Top 10% in Molecular Biology
Koh‐ichi Nagata

What does Koh‐ichi Nagata research?

Koh‐ichi Nagata's work is mostly in Biochemistry, Genetics and Molecular Biology (57%), Neuroscience (11%), Medicine (3%).

Life Sciences: 155 works · 68.5% of their workBiochemistry, Genetics and Molecular Biology: 129 works · 57.1% of their workMolecular Biology: 76 works · 33.5% of their workCell Biology: 29 works · 12.9% of their workGenetics: 24 works · 10.7% of their workNeuroscience: 26 works · 11.4% of their workCellular and Molecular Neuroscience: 20 works · 8.7% of their workDevelopmental Neuroscience: 6 works · 2.7% of their workEverything else: 65 works · 28.8% of their workHealth Sciences: 6 works · 2.7% of their workMedicine: 6 works · 2.7% of their workSurgery: 6 works · 2.7% of their workTHEIR WORKKoh‐ichi Nagata226works
Life Sciences68%Health Sciences3%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology12957.1%
    2. Neuroscience2611.4%
  2. Everything else6528.8%
    1. Medicine62.7%
  1. 1Molecular Biology34% of their works
  2. 2Cell Biology13% of their works
  3. 3Genetics11% of their works
  4. 4Cellular and Molecular Neuroscience9% of their works
  5. 5Surgery3% of their works
  6. 6Developmental Neuroscience3% of their works

Research topics

Which keywords describe Koh‐ichi Nagata's research?

The keywords of their largest research topics: Dendritic Spines, Cancer Therapy, Brain Development, Fragile X Syndrome, Rett Syndrome, Protein Kinases, Ras Signaling and Endocytosis.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Dendritic Spines522 topics
  2. Cancer Therapy492 topics
  3. Brain Development482 topics
  4. Fragile X Syndrome321 topic
  5. Rett Syndrome321 topic
  6. Protein Kinases311 topic
  7. Ras Signaling311 topic
  8. Endocytosis221 topic
  9. Membrane Fusion221 topic
  10. Neurotransmission201 topic
  11. Synaptic Plasticity201 topic
  12. Cell Division181 topic
  13. Microtubules181 topic
  14. Adult Stem Cells161 topic
  15. Neurogenesis161 topic
  16. Cardiomyocyte Renewal141 topic
  17. Heart Regeneration141 topic
  18. Autism Spectrum Disorders131 topic
  19. Genetics131 topic

Where does Koh‐ichi Nagata work?

Koh‐ichi Nagata's main affiliation in the publication record is Aichi Human Service Center, Japan.

How many publications and citations does Koh‐ichi Nagata have?

OpenAlex counts 226 works and 4,255 citations for Koh‐ichi Nagata, an h-index of 35, ranking #5,280,356 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Other researchers named Koh‐ichi Nagata

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Koh‐ichi Nagata. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.