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Researcher · Radboud University Nijmegen · Netherlands

Martin W.I.M. Horstink

Martin W.I.M. Horstink publishes mostly in Neurology, Cellular and Molecular Neuroscience and Cognitive Neuroscience, on topics such as Parkinson's Disease Mechanisms and Treatments, Neurological disorders and treatments and Neurological diseases and metabolism.

OpenAlex
World rank
#1,092,717
of 7,710,434 ranked researchers
Rank in Netherlands
#25,306
of 107,149
Works
131
Citations
7.8k
Citations per work
59.6
h-index
43
i10-index 90Top 4.9% in Neurology (Medicine)
Martin W.I.M. Horstink

What does Martin W.I.M. Horstink research?

Martin W.I.M. Horstink's work is mostly in Medicine (44%), Neuroscience (27%), Engineering (4%).

Health Sciences: 58 works · 44.4% of their workMedicine: 58 works · 44.4% of their workNeurology: 53 works · 40.8% of their workPsychiatry and Mental health: 5 works · 3.6% of their workLife Sciences: 36 works · 27.2% of their workNeuroscience: 36 works · 27.2% of their workCellular and Molecular Neuroscience: 13 works · 9.9% of their workCognitive Neuroscience: 13 works · 9.6% of their workNeurology: 10 works · 7.7% of their workEverything else: 32 works · 24.8% of their workPhysical Sciences: 5 works · 3.6% of their workEngineering: 5 works · 3.6% of their workBiomedical Engineering: 5 works · 3.6% of their workTHEIR WORKMartin W.I.M.Horstink131works
Life Sciences27%Physical Sciences4%Health Sciences44%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine5844.4%
    1. Neuroscience3627.2%
  3. Everything else3224.8%
    1. Engineering53.6%
  1. 1Neurology41% of their works
  2. 2Cellular and Molecular Neuroscience10% of their works
  3. 3Cognitive Neuroscience10% of their works
  4. 4Neurology8% of their works
  5. 5Psychiatry and Mental health4% of their works
  6. 6Biomedical Engineering4% of their works

Research topics

Which keywords describe Martin W.I.M. Horstink's research?

The keywords of their largest research topics: Parkinson's Disease, Mitochondrial Dysfunction, Neurodegeneration, Dopaminergic Neurons, a-Synuclein, Genetic Risk Factors, Neurological Disorders and Deep Brain Stimulation.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Parkinson's Disease1363 topics
  2. Mitochondrial Dysfunction1013 topics
  3. Neurodegeneration1003 topics
  4. Dopaminergic Neurons852 topics
  5. Genetic Risk Factors751 topic
  6. a-Synuclein751 topic
  7. Neurological Disorders642 topics
  8. Deep Brain Stimulation511 topic
  9. Subthalamic Nucleus511 topic
  10. Brain Iron Accumulation151 topic
  11. Pantothenate Kinase151 topic
  12. Botulinum Toxin131 topic
  13. Electromyography131 topic
  14. Muscle Fatigue131 topic
  15. Neurotoxins131 topic
  16. Huntington's Disease111 topic
  17. Protein Aggregation111 topic
  18. NR4A2101 topic
  19. Nurr1101 topic
  20. Brain-Computer Interfaces91 topic
  21. EEG Analysis91 topic

Where does Martin W.I.M. Horstink work?

Martin W.I.M. Horstink's main affiliation in the publication record is Radboud University Nijmegen, Netherlands.

How many publications and citations does Martin W.I.M. Horstink have?

OpenAlex counts 131 works and 7,807 citations for Martin W.I.M. Horstink, an h-index of 43, ranking #1,092,717 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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