Science Explorer Back to mapMap
Discussion
Researcher · Bharathidasan University · India

Mahesh Kandasamy

Mahesh Kandasamy publishes mostly in Molecular Biology, Cellular and Molecular Neuroscience and Neurology, on topics such as Neurogenesis and neuroplasticity mechanisms, Genetic Neurodegenerative Diseases and Botulinum Toxin and Related Neurological Disorders.

ORCID 0000-0002-6720-9082 OpenAlex
World rank
#1,821,232
of 7,710,434 ranked researchers
Rank in India
#30,488
of 280,097
Works
86
Citations
2.1k
Citations per work
24.7
h-index
30
i10-index 44Top 15% in Molecular Biology
Mahesh Kandasamy

What does Mahesh Kandasamy research?

Mahesh Kandasamy's work is mostly in Neuroscience (32%), Medicine (18%), Biochemistry, Genetics and Molecular Biology (14%).

Life Sciences: 40 works · 46.8% of their workNeuroscience: 28 works · 32.5% of their workCellular and Molecular Neuroscience: 12 works · 13.9% of their workDevelopmental Neuroscience: 11 works · 12.3% of their workNeurology: 5 works · 6.3% of their workBiochemistry, Genetics and Molecular Biology: 12 works · 14.3% of their workMolecular Biology: 12 works · 14.3% of their workEverything else: 30 works · 34.9% of their workHealth Sciences: 16 works · 18.3% of their workMedicine: 16 works · 18.3% of their workNeurology: 12 works · 13.5% of their workPhysiology: 4 works · 4.8% of their workTHEIR WORKMahesh Kandasamy86works
Life Sciences47%Health Sciences18%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience2832.5%
    2. Biochemistry, Genetics and Molecular Biology1214.3%
  2. Everything else3034.9%
    1. Medicine1618.3%
  1. 1Molecular Biology14% of their works
  2. 2Cellular and Molecular Neuroscience14% of their works
  3. 3Neurology14% of their works
  4. 4Developmental Neuroscience12% of their works
  5. 5Neurology6% of their works
  6. 6Physiology5% of their works

Research topics

Which keywords describe Mahesh Kandasamy's research?

The keywords of their largest research topics: Brain Development, Mitochondrial Dysfunction, Adult Stem Cells, Neurogenesis, Neurodegeneration, Oxidative Stress, Neurological Disorders and Huntington's Disease.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 23 words, with their numbers
  1. Brain Development402 topics
  2. Mitochondrial Dysfunction313 topics
  3. Adult Stem Cells301 topic
  4. Neurogenesis301 topic
  5. Neurodegeneration233 topics
  6. Oxidative Stress213 topics
  7. Neurological Disorders182 topics
  8. Huntington's Disease161 topic
  9. Neuroinflammation162 topics
  10. Protein Aggregation161 topic
  11. Parkinson's Disease142 topics
  12. Botulinum Toxin111 topic
  13. Neurotoxins111 topic
  14. Astrocytes101 topic
  15. Microglia101 topic
  16. Mitochondria81 topic
  17. Reactive Oxygen Species81 topic
  18. Deep Brain Stimulation71 topic
  19. Dopaminergic Neurons71 topic
  20. Subthalamic Nucleus71 topic
  21. a-Synuclein71 topic
  22. Amyloid Hypothesis61 topic
  23. Tau Pathology61 topic

Where does Mahesh Kandasamy work?

Mahesh Kandasamy's main affiliation in the publication record is Bharathidasan University, India.

How many publications and citations does Mahesh Kandasamy have?

OpenAlex counts 86 works and 2,125 citations for Mahesh Kandasamy, an h-index of 30, ranking #1,821,232 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Mahesh Kandasamy. Posts are plain text and can be reported.