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Researcher · Fox Chase Chemical Diversity Center · United States

Mark E. McDonnell

Mark E. McDonnell publishes mostly in Organic Chemistry, Molecular Biology and Cellular and Molecular Neuroscience, on topics such as Pain Mechanisms and Treatments, Ion Channels and Receptors and Pharmacological Receptor Mechanisms and Effects.

ORCID 0000-0002-9972-5217 OpenAlex
World rank
#3,763,914
of 7,710,434 ranked researchers
Rank in United States
#1,038,970
of 1,627,098
Works
49
Citations
826
Citations per work
16.9
h-index
16
i10-index 22Top 33% in Organic Chemistry
Mark E. McDonnell

What does Mark E. McDonnell research?

Mark E. McDonnell's work is mostly in Chemistry (24%), Biochemistry, Genetics and Molecular Biology (18%), Medicine (17%).

Life Sciences: 15 works · 30.4% of their workBiochemistry, Genetics and Molecular Biology: 9 works · 18.1% of their workMolecular Biology: 9 works · 18.1% of their workNeuroscience: 6 works · 12.3% of their workCellular and Molecular Neuroscience: 6 works · 12.3% of their workEverything else: 14 works · 29.0% of their workPhysical Sciences: 12 works · 23.9% of their workChemistry: 12 works · 23.9% of their workOrganic Chemistry: 12 works · 23.9% of their workHealth Sciences: 8 works · 16.7% of their workMedicine: 8 works · 16.7% of their workOncology: 3 works · 5.8% of their workPhysiology: 3 works · 5.8% of their workPharmacology: 2 works · 5.1% of their workTHEIR WORKMark E. McDonnell49works
Life Sciences30%Physical Sciences24%Health Sciences17%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology918.1%
    2. Neuroscience612.3%
  2. Everything else1429.0%
    1. Chemistry1223.9%
    1. Medicine816.7%
  1. 1Organic Chemistry24% of their works
  2. 2Molecular Biology18% of their works
  3. 3Cellular and Molecular Neuroscience12% of their works
  4. 4Oncology6% of their works
  5. 5Physiology6% of their works
  6. 6Pharmacology5% of their works

Research topics

Which keywords describe Mark E. McDonnell's research?

The keywords of their largest research topics: Nociception, Calcium Signaling, Natural Product Synthesis, Central Sensitization, Enantioselective Synthesis, Neuropathic Pain, Ion Channels and TRP Channels.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 20 words, with their numbers
  1. Nociception142 topics
  2. Calcium Signaling112 topics
  3. Natural Product Synthesis92 topics
  4. Central Sensitization81 topic
  5. Enantioselective Synthesis82 topics
  6. Neuropathic Pain81 topic
  7. Ion Channels61 topic
  8. TRP Channels61 topic
  9. Chaperone51 topic
  10. Neurotransmission51 topic
  11. Sigma-1 Receptor51 topic
  12. Synaptic Plasticity51 topic
  13. Wittig reaction51 topic
  14. stabilized ylides51 topic
  15. Neuropeptides41 topic
  16. Olefin Metathesis41 topic
  17. Organophosphorus Synthesis41 topic
  18. Phosphonate Chemistry41 topic
  19. Receptors41 topic
  20. Ruthenium Catalysts41 topic

Where does Mark E. McDonnell work?

Mark E. McDonnell's main affiliation in the publication record is Fox Chase Chemical Diversity Center, United States.

How many publications and citations does Mark E. McDonnell have?

OpenAlex counts 49 works and 826 citations for Mark E. McDonnell, an h-index of 16, ranking #3,763,914 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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