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Researcher · Amgen (United States) · United States

Martin Citron

Martin Citron publishes mostly in Physiology, Molecular Biology and Computational Theory and Mathematics, on topics such as Alzheimer's disease research and treatments, Computational Drug Discovery Methods and Cholinesterase and Neurodegenerative Diseases.

ORCID 0000-0001-6893-8204 OpenAlex
World rank
#73,506
of 7,710,434 ranked researchers
Rank in United States
#28,373
of 1,627,098
Works
123
Citations
26k
Citations per work
214.2
h-index
56
i10-index 82Top 2.7% in Physiology (Medicine)
Martin Citron

What does Martin Citron research?

Martin Citron's work is mostly in Medicine (46%), Biochemistry, Genetics and Molecular Biology (18%), Computer Science (8%).

Health Sciences: 57 works · 46.1% of their workMedicine: 57 works · 46.1% of their workPhysiology: 30 works · 24.5% of their workNeurology: 9 works · 7.7% of their workPharmacology: 9 works · 7.4% of their workOncology: 8 works · 6.5% of their workEverything else: 34 works · 27.3% of their workLife Sciences: 23 works · 18.3% of their workBiochemistry, Genetics and Molecular Biology: 23 works · 18.3% of their workMolecular Biology: 23 works · 18.3% of their workPhysical Sciences: 10 works · 8.3% of their workComputer Science: 10 works · 8.3% of their workComputational Theory and Mathematics: 10 works · 8.3% of their workTHEIR WORKMartin Citron123works
Life Sciences18%Physical Sciences8%Health Sciences46%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine5746.1%
  2. Everything else3427.3%
    1. Biochemistry, Genetics and Molecular Biology2318.3%
    1. Computer Science108.3%
  1. 1Physiology24% of their works
  2. 2Molecular Biology18% of their works
  3. 3Computational Theory and Mathematics8% of their works
  4. 4Neurology8% of their works
  5. 5Pharmacology7% of their works
  6. 6Oncology6% of their works

Research topics

Which keywords describe Martin Citron's research?

The keywords of their largest research topics: Neurodegeneration, Oxidative Stress, Amyloid Hypothesis, Tau Pathology, Neurodegenerative Diseases, Molecular Docking, Nuclear Receptors and Virtual Screening.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Neurodegeneration1083 topics
  2. Oxidative Stress992 topics
  3. Amyloid Hypothesis821 topic
  4. Tau Pathology821 topic
  5. Neurodegenerative Diseases392 topics
  6. Molecular Docking281 topic
  7. Nuclear Receptors282 topics
  8. Virtual Screening281 topic
  9. Dopaminergic Neurons262 topics
  10. Parkinson's Disease262 topics
  11. Acetylcholinesterase251 topic
  12. Cholinesterase Inhibitors251 topic
  13. ATP-dependent Transporters191 topic
  14. Multidrug Resistance191 topic
  15. Genetic Risk Factors171 topic
  16. a-Synuclein171 topic
  17. Cardiac Amyloidosis161 topic
  18. Transthyretin161 topic
  19. Molecular Basis141 topic
  20. Prion Diseases141 topic
  21. NR4A291 topic
  22. Nurr191 topic

Where does Martin Citron work?

Martin Citron's main affiliation in the publication record is Amgen (United States), United States.

How many publications and citations does Martin Citron have?

OpenAlex counts 123 works and 26,345 citations for Martin Citron, an h-index of 56, ranking #73,506 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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