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Researcher · Max Rubner Institut · Germany

Martin Klempt

Martin Klempt publishes mostly in Molecular Biology, Endocrinology, Diabetes and Metabolism and Health, Toxicology and Mutagenesis, on topics such as Growth Hormone and Insulin-like Growth Factors, S100 Proteins and Annexins and Protein Hydrolysis and Bioactive Peptides.

ORCID 0000-0001-7312-8468 OpenAlex
World rank
#1,903,177
of 7,710,434 ranked researchers
Rank in Germany
#93,964
of 364,343
Works
67
Citations
3k
Citations per work
44.4
h-index
25
i10-index 39Top 22% in Molecular Biology
Martin Klempt

What does Martin Klempt research?

Martin Klempt's work is mostly in Biochemistry, Genetics and Molecular Biology (23%), Medicine (11%), Environmental Science (6%).

Everything else: 37 works · 54.9% of their workLife Sciences: 19 works · 27.7% of their workBiochemistry, Genetics and Molecular Biology: 16 works · 23.4% of their workMolecular Biology: 12 works · 17.4% of their workCancer Research: 4 works · 6.0% of their workImmunology and Microbiology: 3 works · 4.3% of their workImmunology: 3 works · 4.3% of their workHealth Sciences: 8 works · 11.4% of their workMedicine: 8 works · 11.4% of their workEndocrinology, Diabetes and Metabolism: 4 works · 6.5% of their workPediatrics, Perinatology and Child Health: 3 works · 4.9% of their workPhysical Sciences: 4 works · 6.0% of their workEnvironmental Science: 4 works · 6.0% of their workHealth, Toxicology and Mutagenesis: 4 works · 6.0% of their workTHEIR WORKMartin Klempt67works
Life Sciences28%Physical Sciences6%Health Sciences11%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else3754.9%
    1. Biochemistry, Genetics and Molecular Biology1623.4%
    2. Immunology and Microbiology34.3%
    1. Medicine811.4%
    1. Environmental Science46.0%
  1. 1Molecular Biology17% of their works
  2. 2Endocrinology, Diabetes and Metabolism6% of their works
  3. 3Health, Toxicology and Mutagenesis6% of their works
  4. 4Cancer Research6% of their works
  5. 5Pediatrics, Perinatology and Child Health5% of their works
  6. 6Immunology4% of their works

Research topics

Which keywords describe Martin Klempt's research?

The keywords of their largest research topics: Neuroendocrine Control, IGF-1, Insulin-like Growth Factors, Inflammation, S100 Proteins, Antioxidant Activity, Bioactive Peptides and GPR54 Gene.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 17 words, with their numbers
  1. Neuroendocrine Control192 topics
  2. IGF-1121 topic
  3. Insulin-like Growth Factors121 topic
  4. Inflammation81 topic
  5. S100 Proteins81 topic
  6. Antioxidant Activity71 topic
  7. Bioactive Peptides71 topic
  8. GPR54 Gene71 topic
  9. Puberty71 topic
  10. Bronchopulmonary Dysplasia61 topic
  11. Preterm Infants61 topic
  12. Brominated Flame Retardants51 topic
  13. Early-Life Conditions51 topic
  14. Fetal Programming51 topic
  15. Polycyclic Aromatic Hydrocarbons51 topic
  16. Prebiotics51 topic
  17. Probiotics51 topic

Where does Martin Klempt work?

Martin Klempt's main affiliation in the publication record is Max Rubner Institut, Germany.

How many publications and citations does Martin Klempt have?

OpenAlex counts 67 works and 2,978 citations for Martin Klempt, an h-index of 25, ranking #1,903,177 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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