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Researcher · Salk Institute for Biological Studies · United States

Nawab John Dar

Nawab John Dar publishes mostly in Molecular Biology, Complementary and alternative medicine and Physiology, on topics such as Mitochondrial Function and Pathology, Ferroptosis and cancer prognosis and Neuroinflammation and Neurodegeneration Mechanisms.

ORCID 0000-0003-2021-0665 OpenAlex
World rank
#478,774
of 7,710,434 ranked researchers
Rank in United States
#167,380
of 1,627,098
Works
53
Citations
1.4k
Citations per work
26.5
h-index
16
i10-index 24Top 46% in Molecular Biology
Nawab John Dar

What does Nawab John Dar research?

Nawab John Dar's work is mostly in Medicine (28%), Biochemistry, Genetics and Molecular Biology (22%), Neuroscience (13%).

Everything else: 19 works · 36.5% of their workLife Sciences: 19 works · 35.8% of their workBiochemistry, Genetics and Molecular Biology: 12 works · 22.3% of their workMolecular Biology: 12 works · 22.3% of their workNeuroscience: 7 works · 13.5% of their workCellular and Molecular Neuroscience: 4 works · 7.4% of their workNeurology: 3 works · 6.1% of their workHealth Sciences: 15 works · 27.7% of their workMedicine: 15 works · 27.7% of their workComplementary and alternative medicine: 6 works · 10.8% of their workPhysiology: 5 works · 10.1% of their workPulmonary and Respiratory Medicine: 4 works · 6.8% of their workTHEIR WORKNawab John Dar53works
Life Sciences36%Health Sciences28%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else1936.5%
    1. Biochemistry, Genetics and Molecular Biology1222.3%
    2. Neuroscience713.5%
    1. Medicine1527.7%
  1. 1Molecular Biology22% of their works
  2. 2Complementary and alternative medicine11% of their works
  3. 3Physiology10% of their works
  4. 4Cellular and Molecular Neuroscience7% of their works
  5. 5Pulmonary and Respiratory Medicine7% of their works
  6. 6Neurology6% of their works

Research topics

Which keywords describe Nawab John Dar's research?

The keywords of their largest research topics: Oxidative Stress, Cell Death, Cancer, Neuroprotective, Neurodegeneration, Neuroinflammation, Brain Development and Ferroptosis.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Oxidative Stress223 topics
  2. Cell Death162 topics
  3. Cancer152 topics
  4. Neuroprotective142 topics
  5. Neurodegeneration132 topics
  6. Neuroinflammation132 topics
  7. Brain Development102 topics
  8. Ferroptosis81 topic
  9. Mitochondria81 topic
  10. Reactive Oxygen Species81 topic
  11. Tumor Microenvironment81 topic
  12. Astrocytes71 topic
  13. Bacopa monnieri71 topic
  14. Centella asiatica71 topic
  15. Microglia71 topic
  16. Ocimum sanctum71 topic
  17. Withania somnifera71 topic
  18. Amyloid Hypothesis61 topic
  19. Tau Pathology61 topic
  20. Neurotransmission41 topic
  21. Synaptic Plasticity41 topic
  22. Adult Stem Cells31 topic
  23. Neurogenesis31 topic

Where does Nawab John Dar work?

Nawab John Dar's main affiliation in the publication record is Salk Institute for Biological Studies, United States.

How many publications and citations does Nawab John Dar have?

OpenAlex counts 53 works and 1,404 citations for Nawab John Dar, an h-index of 16, ranking #478,774 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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