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Researcher · Defence Science and Technology Laboratory · United Kingdom

Richard W. Titball

Richard W. Titball publishes mostly in Molecular Biology, Genetics and Infectious Diseases, on topics such as Yersinia bacterium, plague, ectoparasites research, Bacillus and Francisella bacterial research and Clostridium difficile and Clostridium perfringens research.

ORCID 0000-0002-0162-2077 OpenAlex
World rank
#190,170
of 7,710,434 ranked researchers
Rank in United Kingdom
#17,300
of 367,258
Works
439
Citations
25k
Citations per work
57.7
h-index
82
i10-index 307Top 0.6% in Molecular Biology
Richard W. Titball

What does Richard W. Titball research?

Richard W. Titball's work is mostly in Biochemistry, Genetics and Molecular Biology (35%), Medicine (20%), Immunology and Microbiology (9%).

Life Sciences: 190 works · 43.2% of their workBiochemistry, Genetics and Molecular Biology: 151 works · 34.5% of their workMolecular Biology: 72 works · 16.5% of their workGenetics: 58 works · 13.2% of their workEndocrinology: 21 works · 4.8% of their workImmunology and Microbiology: 38 works · 8.7% of their workImmunology: 38 works · 8.7% of their workEverything else: 163 works · 37.2% of their workHealth Sciences: 86 works · 19.6% of their workMedicine: 86 works · 19.6% of their workInfectious Diseases: 51 works · 11.7% of their workEpidemiology: 35 works · 7.9% of their workTHEIR WORKRichard W.Titball439works
Life Sciences43%Health Sciences20%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology15134.5%
    2. Immunology and Microbiology388.7%
  2. Everything else16337.2%
    1. Medicine8619.6%
  1. 1Molecular Biology16% of their works
  2. 2Genetics13% of their works
  3. 3Infectious Diseases12% of their works
  4. 4Immunology9% of their works
  5. 5Epidemiology8% of their works
  6. 6Endocrinology5% of their works

Research topics

Which keywords describe Richard W. Titball's research?

The keywords of their largest research topics: Epidemiology, Type III Secretion System, Pathogenesis, Evolution, Genomics, Bacterial Pathogenesis, Antibiotic Resistance and plague.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Epidemiology1983 topics
  2. Type III Secretion System1782 topics
  3. Pathogenesis1732 topics
  4. Evolution1402 topics
  5. Genomics1402 topics
  6. Bacterial Pathogenesis1372 topics
  7. Antibiotic Resistance1212 topics
  8. Yersinia pestis991 topic
  9. plague991 topic
  10. Anthrax941 topic
  11. Tularemia941 topic
  12. Antibiotic-Associated Diarrhea811 topic
  13. Fecal Microbiota Transplantation811 topic
  14. Burkholderia pseudomallei791 topic
  15. Melioidosis791 topic
  16. Immunotoxin481 topic
  17. Ricin Poisoning481 topic
  18. Bacterial Physiology421 topic
  19. Gene Regulation421 topic
  20. Ecology411 topic
  21. Virus411 topic
  22. Cholera381 topic
  23. Vibrio cholerae381 topic

Where does Richard W. Titball work?

Richard W. Titball's main affiliation in the publication record is Defence Science and Technology Laboratory, United Kingdom.

How many publications and citations does Richard W. Titball have?

OpenAlex counts 439 works and 25,326 citations for Richard W. Titball, an h-index of 82, ranking #190,170 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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