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Researcher · Merz (Germany) · Germany

Tadeusz Frankiewicz

Tadeusz Frankiewicz publishes mostly in Cellular and Molecular Neuroscience, Molecular Biology and Neurology, on topics such as Neuroscience and Neuropharmacology Research, Ion channel regulation and function and Pharmacological Receptor Mechanisms and Effects.

OpenAlex
World rank
#3,613,898
of 7,710,434 ranked researchers
Rank in Germany
#180,816
of 364,343
Works
18
Citations
677
Citations per work
37.6
h-index
7
i10-index 7Top 92% in Cellular and Molecular Neuroscience
Tadeusz Frankiewicz

What does Tadeusz Frankiewicz research?

Tadeusz Frankiewicz's work is mostly in Neuroscience (46%), Biochemistry, Genetics and Molecular Biology (18%), Medicine (9%).

Life Sciences: 11 works · 63.7% of their workNeuroscience: 8 works · 45.5% of their workCellular and Molecular Neuroscience: 6 works · 34.1% of their workNeurology: 2 works · 11.4% of their workBiochemistry, Genetics and Molecular Biology: 3 works · 18.2% of their workMolecular Biology: 3 works · 18.2% of their workEverything else: 4 works · 22.8% of their workHealth Sciences: 2 works · 9.0% of their workMedicine: 2 works · 9.0% of their workPediatrics, Perinatology and Child Health: 1 works · 4.5% of their workPsychiatry and Mental health: 1 works · 4.5% of their workPhysical Sciences: 1 works · 4.5% of their workChemistry: 1 works · 4.5% of their workSpectroscopy: 1 works · 4.5% of their workTHEIR WORKTadeuszFrankiewicz18works
Life Sciences64%Physical Sciences4%Health Sciences9%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience845.5%
    2. Biochemistry, Genetics and Molecular Biology318.2%
  2. Everything else422.8%
    1. Medicine29.0%
    1. Chemistry14.5%
  1. 1Cellular and Molecular Neuroscience34% of their works
  2. 2Molecular Biology18% of their works
  3. 3Neurology11% of their works
  4. 4Pediatrics, Perinatology and Child Health4% of their works
  5. 5Spectroscopy4% of their works
  6. 6Psychiatry and Mental health4% of their works

Research topics

Which keywords describe Tadeusz Frankiewicz's research?

The keywords of their largest research topics: Neurotransmission, Synaptic Plasticity, Neurodegenerative Diseases, Oxidative Stress, Ion Channels, Voltage-Gated Channels, Antiepileptic Drugs and Cerebral Hypoperfusion.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Neurotransmission131 topic
  2. Synaptic Plasticity131 topic
  3. Neurodegenerative Diseases63 topics
  4. Oxidative Stress42 topics
  5. Ion Channels31 topic
  6. Voltage-Gated Channels31 topic
  7. Antiepileptic Drugs21 topic
  8. Cerebral Hypoperfusion21 topic
  9. Chaperone21 topic
  10. Fluorescent Chemosensors21 topic
  11. Hippocampus21 topic
  12. Ion Detection21 topic
  13. Memory21 topic
  14. Microglia21 topic
  15. Neuroinflammation21 topic
  16. Prenatal Exposure21 topic
  17. Sigma-1 Receptor21 topic
  18. Vascular Cognitive Impairment21 topic

Where does Tadeusz Frankiewicz work?

Tadeusz Frankiewicz's main affiliation in the publication record is Merz (Germany), Germany.

How many publications and citations does Tadeusz Frankiewicz have?

OpenAlex counts 18 works and 677 citations for Tadeusz Frankiewicz, an h-index of 7, ranking #3,613,898 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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