Alexei Verkhratsky
Alexei Verkhratsky publishes mostly in Cellular and Molecular Neuroscience, Molecular Biology and Neurology, on topics such as Neuroscience and Neuropharmacology Research, Neuroinflammation and Neurodegeneration Mechanisms and Alzheimer's disease research and treatments.
- World rank
- #37,010 of 7,710,434 ranked researchers
- Rank in United Kingdom
- #2,877 of 367,258
- Works
- 809
- Citations
- 68k
- Citations per work
- 84.0
What does Alexei Verkhratsky research?
Alexei Verkhratsky's work is mostly in Neuroscience (40%), Biochemistry, Genetics and Molecular Biology (21%), Medicine (7%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Life Sciences49861.6%
- Neuroscience32540.2%
- Biochemistry, Genetics and Molecular Biology17321.4%
- Everything else25131.0%
- Health Sciences607.4%
- Medicine607.4%
- 1Cellular and Molecular Neuroscience20% of their works
- 2Molecular Biology16% of their works
- 3Neurology15% of their works
- 4Physiology7% of their works
- 5Developmental Neuroscience6% of their works
- 6Physiology6% of their works
Research topics
- Neuroscience and Neuropharmacology Research
- Neuroinflammation and Neurodegeneration Mechanisms
- Alzheimer's disease research and treatments
- Neurogenesis and neuroplasticity mechanisms
- Ion channel regulation and function
- Adenosine and Purinergic Signaling
- Mitochondrial Function and Pathology
- Cellular transport and secretion
Which keywords describe Alexei Verkhratsky's research?
The keywords of their largest research topics: Neurodegeneration, Neuroinflammation, Brain Development, Neurotransmission, Synaptic Plasticity, Astrocytes, Microglia and Oxidative Stress.
- Membrane Fusion
- Reactive Oxygen Species
- Purinergic Signalling
- Voltage-Gated Channels
- Neurogenesis
- Tau Pathology
- Oxidative Stress
- Astrocytes
- Neurotransmission
- Neuroinflammation
- Neurodegeneration
- Brain Development
- Synaptic Plasticity
- Microglia
- Amyloid Hypothesis
- Adult Stem Cells
- Ion Channels
- Adenosine Receptors
- Mitochondria
- Endocytosis
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 20 words, with their numbers
- Neurodegeneration3832 topics
- Neuroinflammation3832 topics
- Brain Development3822 topics
- Neurotransmission3081 topic
- Synaptic Plasticity3081 topic
- Astrocytes2631 topic
- Microglia2631 topic
- Oxidative Stress1622 topics
- Amyloid Hypothesis1201 topic
- Tau Pathology1201 topic
- Adult Stem Cells1191 topic
- Neurogenesis1191 topic
- Ion Channels1111 topic
- Voltage-Gated Channels1111 topic
- Adenosine Receptors861 topic
- Purinergic Signalling861 topic
- Mitochondria421 topic
- Reactive Oxygen Species421 topic
- Endocytosis401 topic
- Membrane Fusion401 topic
Where does Alexei Verkhratsky work?
Alexei Verkhratsky's main affiliation in the publication record is University of Manchester, United Kingdom.
How many publications and citations does Alexei Verkhratsky have?
OpenAlex counts 809 works and 67,937 citations for Alexei Verkhratsky, an h-index of 122, ranking #37,010 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
Discussion
Ask a question or share a note about Alexei Verkhratsky. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.