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Researcher · Reata Pharmaceuticals (United States) · United States

Angie Goldsberry

Angie Goldsberry publishes mostly in Molecular Biology, Nephrology and Cellular and Molecular Neuroscience, on topics such as Genetic Neurodegenerative Diseases, Cell Adhesion Molecules Research and Chronic Kidney Disease and Diabetes.

ORCID 0000-0003-4143-2195 OpenAlex
World rank
#1,430,963
of 7,710,434 ranked researchers
Rank in United States
#450,942
of 1,627,098
Works
46
Citations
2.2k
Citations per work
46.8
h-index
13
i10-index 13Top 59% in Molecular Biology
Angie Goldsberry

What does Angie Goldsberry research?

Angie Goldsberry's work is mostly in Medicine (28%), Biochemistry, Genetics and Molecular Biology (17%), Neuroscience (8%).

Everything else: 22 works · 46.8% of their workHealth Sciences: 13 works · 27.5% of their workMedicine: 13 works · 27.5% of their workNephrology: 4 works · 8.3% of their workCardiology and Cardiovascular Medicine: 3 works · 6.4% of their workImmunology and Allergy: 3 works · 6.4% of their workNeurology: 3 works · 6.4% of their workLife Sciences: 12 works · 25.7% of their workBiochemistry, Genetics and Molecular Biology: 8 works · 17.4% of their workMolecular Biology: 8 works · 17.4% of their workNeuroscience: 4 works · 8.3% of their workCellular and Molecular Neuroscience: 4 works · 8.3% of their workTHEIR WORKAngie Goldsberry46works
Life Sciences26%Health Sciences28%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else2246.8%
    1. Medicine1327.5%
    1. Biochemistry, Genetics and Molecular Biology817.4%
    2. Neuroscience48.3%
  1. 1Molecular Biology17% of their works
  2. 2Nephrology8% of their works
  3. 3Cellular and Molecular Neuroscience8% of their works
  4. 4Immunology and Allergy6% of their works
  5. 5Neurology6% of their works
  6. 6Cardiology and Cardiovascular Medicine6% of their works

Research topics

Which keywords describe Angie Goldsberry's research?

The keywords of their largest research topics: Inflammation, Mitochondrial Dysfunction, Oxidative Stress, Cancer, Angiogenesis, Cardiovascular Disease, Global Burden and Huntington's Disease.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Inflammation153 topics
  2. Mitochondrial Dysfunction142 topics
  3. Oxidative Stress133 topics
  4. Cancer112 topics
  5. Angiogenesis102 topics
  6. Cardiovascular Disease92 topics
  7. Global Burden92 topics
  8. Huntington's Disease91 topic
  9. Protein Aggregation91 topic
  10. Cell Adhesion71 topic
  11. Integrins71 topic
  12. Chronic Kidney Disease61 topic
  13. Glomerular Filtration Rate61 topic
  14. Mitochondria51 topic
  15. Reactive Oxygen Species51 topic
  16. Advanced Glycation End Products41 topic
  17. HMGB141 topic
  18. Keap1-Nrf2-ARE Pathway41 topic
  19. Nrf241 topic
  20. Blood Pressure31 topic
  21. Fibroblast Activation Protein31 topic
  22. Hypertension31 topic
  23. Tumor Microenvironment31 topic

Where does Angie Goldsberry work?

Angie Goldsberry's main affiliation in the publication record is Reata Pharmaceuticals (United States), United States.

How many publications and citations does Angie Goldsberry have?

OpenAlex counts 46 works and 2,153 citations for Angie Goldsberry, an h-index of 13, ranking #1,430,963 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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