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Researcher · University of Groningen · Netherlands

Bart J. L. Eggen

Bart J. L. Eggen publishes mostly in Molecular Biology, Neurology and Immunology, on topics such as Neuroinflammation and Neurodegeneration Mechanisms, Immune cells in cancer and Immune Response and Inflammation.

ORCID 0000-0001-8941-0353 OpenAlex
World rank
#244,620
of 7,710,434 ranked researchers
Rank in Netherlands
#6,220
of 107,149
Works
278
Citations
14k
Citations per work
50.1
h-index
59
i10-index 130Top 2.1% in Molecular Biology
Bart J. L. Eggen

What does Bart J. L. Eggen research?

Bart J. L. Eggen's work is mostly in Neuroscience (21%), Biochemistry, Genetics and Molecular Biology (18%), Immunology and Microbiology (16%).

Life Sciences: 153 works · 55.1% of their workNeuroscience: 59 works · 21.3% of their workNeurology: 49 works · 17.6% of their workDevelopmental Neuroscience: 10 works · 3.7% of their workBiochemistry, Genetics and Molecular Biology: 50 works · 18.0% of their workMolecular Biology: 50 works · 18.0% of their workImmunology and Microbiology: 44 works · 15.8% of their workImmunology: 44 works · 15.8% of their workEverything else: 99 works · 35.5% of their workHealth Sciences: 26 works · 9.4% of their workMedicine: 26 works · 9.4% of their workPhysiology: 16 works · 5.8% of their workSurgery: 10 works · 3.6% of their workTHEIR WORKBart J. L. Eggen278works
Life Sciences55%Health Sciences9%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience5921.3%
    2. Biochemistry, Genetics and Molecular Biology5018.0%
    3. Immunology and Microbiology4415.8%
  2. Everything else9935.5%
    1. Medicine269.4%
  1. 1Molecular Biology18% of their works
  2. 2Neurology18% of their works
  3. 3Immunology16% of their works
  4. 4Physiology6% of their works
  5. 5Developmental Neuroscience4% of their works
  6. 6Surgery4% of their works

Research topics

Which keywords describe Bart J. L. Eggen's research?

The keywords of their largest research topics: Neuroinflammation, Neurodegeneration, Brain Development, Cytokines, Microglia, Astrocytes, Synaptic Pruning and Inflammation.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Neuroinflammation1523 topics
  2. Neurodegeneration1382 topics
  3. Brain Development1372 topics
  4. Cytokines1292 topics
  5. Microglia1292 topics
  6. Astrocytes1151 topic
  7. Synaptic Pruning1151 topic
  8. Inflammation832 topics
  9. Activation541 topic
  10. Macrophage541 topic
  11. Oxidative Stress372 topics
  12. Pattern Recognition Receptors291 topic
  13. Toll-like Receptors291 topic
  14. Amyloid Hypothesis231 topic
  15. Tau Pathology231 topic
  16. Adult Stem Cells221 topic
  17. Neurogenesis221 topic
  18. Depression141 topic
  19. Tryptophan Metabolism141 topic
  20. Single-Cell121 topic
  21. Transcriptomics121 topic
  22. Alternative Splicing111 topic
  23. RNA-Binding Proteins111 topic

Where does Bart J. L. Eggen work?

Bart J. L. Eggen's main affiliation in the publication record is University of Groningen, Netherlands.

How many publications and citations does Bart J. L. Eggen have?

OpenAlex counts 278 works and 13,923 citations for Bart J. L. Eggen, an h-index of 59, ranking #244,620 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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