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Researcher · Universidade Federal de São Paulo · Brazil

Carla Alessandra Scorza

Carla Alessandra Scorza publishes mostly in Neurology, Psychiatry and Mental health and Cellular and Molecular Neuroscience, on topics such as Epilepsy research and treatment, Neuroscience and Neuropharmacology Research and Parkinson's Disease Mechanisms and Treatments.

ORCID 0000-0001-7810-4748 OpenAlex
World rank
#7,557,141
of 7,710,434 ranked researchers
Rank in Brazil
#244,711
of 253,246
Works
387
Citations
2.7k
Citations per work
7.0
h-index
28
i10-index 79Top 13% in Neurology (Medicine)
Carla Alessandra Scorza

What does Carla Alessandra Scorza research?

Carla Alessandra Scorza's work is mostly in Medicine (36%), Neuroscience (8%), Biochemistry, Genetics and Molecular Biology (6%).

Everything else: 194 works · 50.0% of their workHealth Sciences: 139 works · 35.9% of their workMedicine: 139 works · 35.9% of their workNeurology: 70 works · 18.0% of their workPsychiatry and Mental health: 39 works · 10.1% of their workPediatrics, Perinatology and Child Health: 16 works · 4.2% of their workPhysiology: 14 works · 3.6% of their workLife Sciences: 55 works · 14.1% of their workNeuroscience: 33 works · 8.4% of their workCellular and Molecular Neuroscience: 33 works · 8.4% of their workBiochemistry, Genetics and Molecular Biology: 22 works · 5.7% of their workMolecular Biology: 22 works · 5.7% of their workTHEIR WORKCarla AlessandraScorza387works
Life Sciences14%Health Sciences36%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else19450.0%
    1. Medicine13935.9%
    1. Neuroscience338.4%
    2. Biochemistry, Genetics and Molecular Biology225.7%
  1. 1Neurology18% of their works
  2. 2Psychiatry and Mental health10% of their works
  3. 3Cellular and Molecular Neuroscience8% of their works
  4. 4Molecular Biology6% of their works
  5. 5Pediatrics, Perinatology and Child Health4% of their works
  6. 6Physiology4% of their works

Research topics

Which keywords describe Carla Alessandra Scorza's research?

The keywords of their largest research topics: Epilepsy, Antiepileptic Drugs, Oxidative Stress, Neurological Disorders, Classification, Parkinson's Disease, Seizures and Mitochondrial Dysfunction.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 22 words, with their numbers
  1. Epilepsy1122 topics
  2. Antiepileptic Drugs1092 topics
  3. Oxidative Stress983 topics
  4. Neurological Disorders922 topics
  5. Classification811 topic
  6. Parkinson's Disease812 topics
  7. Seizures811 topic
  8. Mitochondrial Dysfunction702 topics
  9. Neurotransmission611 topic
  10. Synaptic Plasticity611 topic
  11. Neurodegeneration501 topic
  12. a-Synuclein501 topic
  13. Neurologic Manifestations381 topic
  14. Post-acute COVID-19 Syndrome381 topic
  15. Deep Brain Stimulation311 topic
  16. Subthalamic Nucleus311 topic
  17. Prenatal Exposure281 topic
  18. Teratogenicity281 topic
  19. Biochemical Genetics241 topic
  20. Metabolic Disorders241 topic
  21. Mitochondria201 topic
  22. Reactive Oxygen Species201 topic

Where does Carla Alessandra Scorza work?

Carla Alessandra Scorza's main affiliation in the publication record is Universidade Federal de São Paulo, Brazil.

How many publications and citations does Carla Alessandra Scorza have?

OpenAlex counts 387 works and 2,725 citations for Carla Alessandra Scorza, an h-index of 28, ranking #7,557,141 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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