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Researcher · University of Puerto Rico at Río Piedras · Puerto Rico

Carlos Alberto Báez-Pagán

Carlos Alberto Báez-Pagán publishes mostly in Molecular Biology, Cell Biology and Neurology, on topics such as Nicotinic Acetylcholine Receptors Study, Ion channel regulation and function and Receptor Mechanisms and Signaling.

ORCID 0000-0002-3590-9893 OpenAlex
World rank
#6,071,494
of 7,710,434 ranked researchers
Rank in Puerto Rico
#1,382
of 1,948
Works
24
Citations
411
Citations per work
17.1
h-index
11
i10-index 12Top 71% in Molecular Biology
Carlos Alberto Báez-Pagán

What does Carlos Alberto Báez-Pagán research?

Carlos Alberto Báez-Pagán's work is mostly in Biochemistry, Genetics and Molecular Biology (69%), Neuroscience (14%), Medicine (4%).

Life Sciences: 20 works · 82.8% of their workBiochemistry, Genetics and Molecular Biology: 16 works · 68.5% of their workMolecular Biology: 15 works · 61.4% of their workCell Biology: 2 works · 7.1% of their workNeuroscience: 3 works · 14.3% of their workCellular and Molecular Neuroscience: 1 works · 5.7% of their workNeurology: 1 works · 5.7% of their workBiological Psychiatry: 1 works · 2.9% of their workEverything else: 3 works · 12.9% of their workHealth Sciences: 1 works · 4.3% of their workMedicine: 1 works · 4.3% of their workPharmacology: 1 works · 4.3% of their workTHEIR WORKCarlos AlbertoBáez-Pagán24works
Life Sciences83%Health Sciences4%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology1668.5%
    2. Neuroscience314.3%
  2. Everything else312.9%
    1. Medicine14.3%
  1. 1Molecular Biology61% of their works
  2. 2Cell Biology7% of their works
  3. 3Neurology6% of their works
  4. 4Cellular and Molecular Neuroscience6% of their works
  5. 5Pharmacology4% of their works
  6. 6Biological Psychiatry3% of their works

Research topics

Which keywords describe Carlos Alberto Báez-Pagán's research?

The keywords of their largest research topics: Crystal Structure, Cell Signaling, Alzheimer's Disease, Neuronal Signaling, Nicotinic Acetylcholine Receptors, Ion Channels, Pharmacology and Voltage-Gated Channels.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Crystal Structure252 topics
  2. Cell Signaling223 topics
  3. Alzheimer's Disease202 topics
  4. Neuronal Signaling171 topic
  5. Nicotinic Acetylcholine Receptors171 topic
  6. Ion Channels111 topic
  7. Pharmacology112 topics
  8. Voltage-Gated Channels111 topic
  9. G-Protein-Coupled Receptors81 topic
  10. Signal Transduction81 topic
  11. Endocytosis52 topics
  12. Acetylcholinesterase31 topic
  13. Caveolae31 topic
  14. Caveolin31 topic
  15. Channelrhodopsin31 topic
  16. Cholinergic Antiinflammatory Pathway31 topic
  17. Cholinesterase Inhibitors31 topic
  18. Optogenetics31 topic
  19. Vagus Nerve Stimulation31 topic
  20. Membrane Fusion21 topic
  21. Rab Proteins21 topic

Where does Carlos Alberto Báez-Pagán work?

Carlos Alberto Báez-Pagán's main affiliation in the publication record is University of Puerto Rico at Río Piedras, Puerto Rico.

How many publications and citations does Carlos Alberto Báez-Pagán have?

OpenAlex counts 24 works and 411 citations for Carlos Alberto Báez-Pagán, an h-index of 11, ranking #6,071,494 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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