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Researcher · UK Dementia Research Institute · United Kingdom

Carola I. Radulescu

Carola I. Radulescu publishes mostly in Cellular and Molecular Neuroscience, Molecular Biology and Cognitive Neuroscience, on topics such as Genetic Neurodegenerative Diseases, Neuroscience and Neuropharmacology Research and Mitochondrial Function and Pathology.

ORCID 0000-0003-4089-9295 OpenAlex
World rank
#1,066,139
of 7,710,434 ranked researchers
Rank in United Kingdom
#85,734
of 367,258
Works
35
Citations
1.1k
Citations per work
30.5
h-index
14
i10-index 18Top 57% in Cellular and Molecular Neuroscience
Carola I. Radulescu

What does Carola I. Radulescu research?

Carola I. Radulescu's work is mostly in Neuroscience (46%), Biochemistry, Genetics and Molecular Biology (23%), Immunology and Microbiology (6%).

Life Sciences: 26 works · 74.8% of their workNeuroscience: 16 works · 46.2% of their workCellular and Molecular Neuroscience: 8 works · 23.1% of their workCognitive Neuroscience: 4 works · 11.0% of their workNeurology: 2 works · 6.6% of their workDevelopmental Neuroscience: 2 works · 5.5% of their workBiochemistry, Genetics and Molecular Biology: 8 works · 23.1% of their workMolecular Biology: 8 works · 23.1% of their workImmunology and Microbiology: 2 works · 5.5% of their workImmunology: 2 works · 5.5% of their workEverything else: 9 works · 25.2% of their workTHEIR WORKCarola I.Radulescu35works
Life Sciences75%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience1646.2%
    2. Biochemistry, Genetics and Molecular Biology823.1%
    3. Immunology and Microbiology25.5%
  2. Everything else925.2%
  1. 1Cellular and Molecular Neuroscience23% of their works
  2. 2Molecular Biology23% of their works
  3. 3Cognitive Neuroscience11% of their works
  4. 4Neurology7% of their works
  5. 5Immunology6% of their works
  6. 6Developmental Neuroscience6% of their works

Research topics

Which keywords describe Carola I. Radulescu's research?

The keywords of their largest research topics: Mitochondrial Dysfunction, Brain Development, Dendritic Spines, Oxidative Stress, Huntington's Disease, Neurodegeneration, Neuroinflammation and Protein Aggregation.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Mitochondrial Dysfunction172 topics
  2. Brain Development143 topics
  3. Dendritic Spines122 topics
  4. Oxidative Stress112 topics
  5. Huntington's Disease101 topic
  6. Neurodegeneration102 topics
  7. Neuroinflammation102 topics
  8. Protein Aggregation101 topic
  9. Neurotransmission91 topic
  10. Synaptic Plasticity91 topic
  11. Mitochondria71 topic
  12. Reactive Oxygen Species71 topic
  13. Astrocytes61 topic
  14. Cortical Networks61 topic
  15. Microglia61 topic
  16. Neuronal Oscillations61 topic
  17. Adult Stem Cells51 topic
  18. Neurogenesis51 topic
  19. Amyloid Hypothesis41 topic
  20. Tau Pathology41 topic
  21. Fragile X Syndrome31 topic
  22. Rett Syndrome31 topic

Where does Carola I. Radulescu work?

Carola I. Radulescu's main affiliation in the publication record is UK Dementia Research Institute, United Kingdom.

How many publications and citations does Carola I. Radulescu have?

OpenAlex counts 35 works and 1,067 citations for Carola I. Radulescu, an h-index of 14, ranking #1,066,139 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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