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Researcher · Sapienza University of Rome · Italy

Cristina Limatola

Cristina Limatola publishes mostly in Molecular Biology, Neurology and Cellular and Molecular Neuroscience, on topics such as Neuroinflammation and Neurodegeneration Mechanisms, Neuroscience and Neuropharmacology Research and Chemokine receptors and signaling.

ORCID 0000-0001-7504-8197 OpenAlex
World rank
#321,811
of 7,710,434 ranked researchers
Rank in Italy
#8,227
of 242,206
Works
265
Citations
10k
Citations per work
38.9
h-index
58
i10-index 156Top 2.3% in Molecular Biology
Cristina Limatola

What does Cristina Limatola research?

Cristina Limatola's work is mostly in Neuroscience (30%), Biochemistry, Genetics and Molecular Biology (17%), Immunology and Microbiology (8%).

Life Sciences: 145 works · 54.6% of their workNeuroscience: 79 works · 29.8% of their workNeurology: 35 works · 13.3% of their workCellular and Molecular Neuroscience: 29 works · 11.1% of their workCognitive Neuroscience: 14 works · 5.4% of their workBiochemistry, Genetics and Molecular Biology: 45 works · 16.9% of their workMolecular Biology: 45 works · 16.9% of their workImmunology and Microbiology: 21 works · 7.9% of their workImmunology: 21 works · 7.9% of their workEverything else: 103 works · 38.7% of their workHealth Sciences: 18 works · 6.7% of their workMedicine: 18 works · 6.7% of their workOncology: 18 works · 6.7% of their workTHEIR WORKCristina Limatola265works
Life Sciences55%Health Sciences7%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience7929.8%
    2. Biochemistry, Genetics and Molecular Biology4516.9%
    3. Immunology and Microbiology217.9%
  2. Everything else10338.7%
    1. Medicine186.7%
  1. 1Molecular Biology17% of their works
  2. 2Neurology13% of their works
  3. 3Cellular and Molecular Neuroscience11% of their works
  4. 4Immunology8% of their works
  5. 5Oncology7% of their works
  6. 6Cognitive Neuroscience5% of their works

Research topics

Which keywords describe Cristina Limatola's research?

The keywords of their largest research topics: Microglia, Neuroinflammation, Brain Development, Astrocytes, Neurodegeneration, Inflammation, Therapeutic Targets and Neurotransmission.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Microglia1052 topics
  2. Neuroinflammation1052 topics
  3. Brain Development982 topics
  4. Astrocytes831 topic
  5. Neurodegeneration831 topic
  6. Inflammation753 topics
  7. Therapeutic Targets582 topics
  8. Neurotransmission501 topic
  9. Synaptic Plasticity501 topic
  10. Cancer Metastasis411 topic
  11. Chemokine Receptors411 topic
  12. Ion Channels241 topic
  13. Voltage-Gated Channels241 topic
  14. Depression221 topic
  15. Tryptophan Metabolism221 topic
  16. Activation171 topic
  17. Adenosine Receptors171 topic
  18. Macrophage171 topic
  19. Purinergic Signalling171 topic
  20. Adult Stem Cells151 topic
  21. Neurogenesis151 topic

Where does Cristina Limatola work?

Cristina Limatola's main affiliation in the publication record is Sapienza University of Rome, Italy.

How many publications and citations does Cristina Limatola have?

OpenAlex counts 265 works and 10,317 citations for Cristina Limatola, an h-index of 58, ranking #321,811 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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