Dietmar Rudolf Thal
Dietmar Rudolf Thal publishes mostly in Physiology, Neurology and Molecular Biology, on topics such as Alzheimer's disease research and treatments, Dementia and Cognitive Impairment Research and Parkinson's Disease Mechanisms and Treatments.
- World rank
- #68,400 of 7,710,434 ranked researchers
- Rank in Belgium
- #371 of 67,186
- Works
- 435
- Citations
- 39k
- Citations per work
- 88.9
What does Dietmar Rudolf Thal research?
Dietmar Rudolf Thal's work is mostly in Medicine (42%), Biochemistry, Genetics and Molecular Biology (13%), Neuroscience (11%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Health Sciences18442.4%
- Medicine18442.4%
- Everything else14533.3%
- Life Sciences10624.3%
- Biochemistry, Genetics and Molecular Biology5813.3%
- Neuroscience4811.0%
- 1Physiology20% of their works
- 2Neurology15% of their works
- 3Molecular Biology13% of their works
- 4Psychiatry and Mental health7% of their works
- 5Neurology7% of their works
- 6Cellular and Molecular Neuroscience4% of their works
Research topics
- Alzheimer's disease research and treatments
- Dementia and Cognitive Impairment Research
- Parkinson's Disease Mechanisms and Treatments
- Amyotrophic Lateral Sclerosis Research
- Neuroinflammation and Neurodegeneration Mechanisms
- Cholinesterase and Neurodegenerative Diseases
- Prion Diseases and Protein Misfolding
- Amyloidosis: Diagnosis, Treatment, Outcomes
Which keywords describe Dietmar Rudolf Thal's research?
The keywords of their largest research topics: Neurodegeneration, Oxidative Stress, Neuroinflammation, Amyloid Hypothesis, Tau Pathology, Alzheimer's Disease, Dementia and Mild Cognitive Impairment.
- Cardiac Amyloidosis
- Molecular Basis
- Acetylcholinesterase
- Astrocytes
- TDP-43
- a-Synuclein
- Mild Cognitive Impairment
- Alzheimer's Disease
- Amyloid Hypothesis
- Oxidative Stress
- Neurodegeneration
- Neuroinflammation
- Tau Pathology
- Dementia
- Parkinson's Disease
- FUS/TLS
- Neurodegenerative Diseases
- Microglia
- Cholinesterase Inhibitors
- Prion Diseases
- Transthyretin
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 21 words, with their numbers
- Neurodegeneration3714 topics
- Oxidative Stress2802 topics
- Neuroinflammation2612 topics
- Amyloid Hypothesis2211 topic
- Tau Pathology2211 topic
- Alzheimer's Disease1052 topics
- Dementia821 topic
- Mild Cognitive Impairment821 topic
- Parkinson's Disease591 topic
- a-Synuclein591 topic
- FUS/TLS511 topic
- TDP-43511 topic
- Neurodegenerative Diseases452 topics
- Astrocytes401 topic
- Microglia401 topic
- Acetylcholinesterase231 topic
- Cholinesterase Inhibitors231 topic
- Molecular Basis221 topic
- Prion Diseases221 topic
- Cardiac Amyloidosis201 topic
- Transthyretin201 topic
Where does Dietmar Rudolf Thal work?
Dietmar Rudolf Thal's main affiliation in the publication record is KU Leuven, Belgium.
How many publications and citations does Dietmar Rudolf Thal have?
OpenAlex counts 435 works and 38,667 citations for Dietmar Rudolf Thal, an h-index of 84, ranking #68,400 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
Discussion
Ask a question or share a note about Dietmar Rudolf Thal. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.