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Researcher · University of California San Diego · United States

Eliezer Masliah

Eliezer Masliah publishes mostly in Physiology, Neurology and Molecular Biology, on topics such as Alzheimer's disease research and treatments, Parkinson's Disease Mechanisms and Treatments and Neuroinflammation and Neurodegeneration Mechanisms.

ORCID 0000-0002-2117-5569 OpenAlex
World rank
#6,439
of 7,710,434 ranked researchers
Rank in United States
#2,223
of 1,627,098
Works
1,235
Citations
183k
Citations per work
148.1
h-index
196
i10-index 902Top 0.1% in Physiology (Medicine)
Eliezer Masliah

What does Eliezer Masliah research?

Eliezer Masliah's work is mostly in Medicine (30%), Neuroscience (20%), Biochemistry, Genetics and Molecular Biology (12%).

Life Sciences: 448 works · 36.3% of their workNeuroscience: 246 works · 19.9% of their workCellular and Molecular Neuroscience: 147 works · 11.9% of their workNeurology: 99 works · 8.0% of their workBiochemistry, Genetics and Molecular Biology: 154 works · 12.5% of their workMolecular Biology: 154 works · 12.5% of their workImmunology and Microbiology: 48 works · 3.9% of their workVirology: 48 works · 3.9% of their workEverything else: 416 works · 33.7% of their workHealth Sciences: 370 works · 30.0% of their workMedicine: 370 works · 30.0% of their workPhysiology: 211 works · 17.1% of their workNeurology: 159 works · 12.9% of their workTHEIR WORKEliezer Masliah1,235works
Life Sciences36%Health Sciences30%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience24619.9%
    2. Biochemistry, Genetics and Molecular Biology15412.5%
    3. Immunology and Microbiology483.9%
  2. Everything else41633.7%
    1. Medicine37030.0%
  1. 1Physiology17% of their works
  2. 2Neurology13% of their works
  3. 3Molecular Biology12% of their works
  4. 4Cellular and Molecular Neuroscience12% of their works
  5. 5Neurology8% of their works
  6. 6Virology4% of their works

Research topics

Which keywords describe Eliezer Masliah's research?

The keywords of their largest research topics: Neurodegeneration, Oxidative Stress, Neuroinflammation, Amyloid Hypothesis, Tau Pathology, Dopaminergic Neurons, Parkinson's Disease and a-Synuclein.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Neurodegeneration1,0944 topics
  2. Oxidative Stress8392 topics
  3. Neuroinflammation6562 topics
  4. Amyloid Hypothesis5221 topic
  5. Tau Pathology5221 topic
  6. Dopaminergic Neurons4382 topics
  7. Parkinson's Disease4382 topics
  8. Genetic Risk Factors3171 topic
  9. a-Synuclein3171 topic
  10. Alzheimer's Disease1812 topics
  11. Astrocytes1341 topic
  12. Microglia1341 topic
  13. AIDS1291 topic
  14. HIV1291 topic
  15. NR4A21211 topic
  16. Nurr11211 topic
  17. Neurotransmission1101 topic
  18. Synaptic Plasticity1101 topic
  19. Acetylcholinesterase921 topic
  20. Cholinesterase Inhibitors921 topic
  21. Dementia891 topic
  22. Mild Cognitive Impairment891 topic

Where does Eliezer Masliah work?

Eliezer Masliah's main affiliation in the publication record is University of California San Diego, United States.

How many publications and citations does Eliezer Masliah have?

OpenAlex counts 1,235 works and 182,899 citations for Eliezer Masliah, an h-index of 196, ranking #6,439 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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